Latest Research in Circular RNAs in diseases
25 research papers · 2026 median publication year
Top Research Topics in Circular RNAs in diseases
- Circular RNAs in diseases — 4 papers
- Cancer, Hypoxia, and Metabolism — 2 papers
- Ubiquitin and proteasome pathways — 2 papers
- Exercise and Physiological Responses — 2 papers
- RNA modifications and cancer — 2 papers
- Hippo pathway signaling and YAP/TAZ — 2 papers
- Protein Degradation and Inhibitors — 2 papers
- Bone and Dental Protein Studies — 1 papers
- Immune cells in cancer — 1 papers
- Protein Kinase Regulation and GTPase Signaling — 1 papers
Highest-Cited Papers
- Circular RNAs in NSCLC: Their Role in Tumorigenesis, Tumor Microenvironment Reshaping, and Response to Therapies
- SelO functions as a tumor suppressor through AMPylating Cdk5rap3
- Single-Cell Transcriptomics Uncovers β-Elemene-Induced Macrophage Reprogramming as a Mechanism to Suppress Osteosarcoma Stemness via the CCL2-Akt Axis
- Shikonin as a Parkin-dependent hypoxia-selective agent against triple-negative breast cancer identified by machine learning
- KDM2B controls HIF levels and activity through its JmjC and CxxC domains.
- SENP7 mediates deSUMOylation and deubiquitination of SP1 to promote lung adenocarcinoma progression and chemoresistance
- Changes in the transcriptome and synthetic lethal dependencies following KRAS mutant expression reveal profound tissue specificity
- CircERBB2 and beyond: a new regulatory layer in HER2-driven cancer
- Exercise-induced systemic and tumor-microenvironment adaptations regulating chemokine-guided immune-cell trafficking
- Application of vibrational spectroscopy in lung cancer diagnosis and treatment: Current status and future directions
- FTO-Driven m6A Demethylation of TNC Promotes Epithelial–Mesenchymal Transition and Gastric Cancer Metastasis
- Transcriptional Regulation by YAP/TAZ via TEADs and Additional Transcription Factors
- E3 Ubiquitin Ligase MDM2 Contributes to Proliferation, Migration/Invasion, Epithelial‐Mesenchymal Transition, and Glycolytic Reprogramming in Lung Cancer via Ubiquitinating and Degrading MYH11 Protein
- Deubiquitination of Vangl by USP6 and USP32 Regulates Planar Cell Polarity Signaling
- Integrative pan-cancer analysis of GAD1 across molecular subtypes, metabolic contexts, and clinical outcomes
- Glucose and Glutamine Deprivation Promotes Breast Cancer Lung Metastasis via BRD4‐Dependent Enhancer Activation
- Cancer‐Associated Fibroblasts Promote Glucose Metabolic Reprogramming and Progression of Triple‐Negative Breast Cancer via Exosomal circFAD104‐Mediated Intercellular Communication
- Restoration of m6A‐Modified circPSMB1 Suppresses Chordoma Progression and Enables Local Nanotherapeutic Intervention
- Guardians of the genome, allies of resistance: Unraveling the role of piRNAs and PIWI proteins in chemotherapy evasion
- Cellular and transcriptomic responses to a PDE4DIP-directed siRNA in MDA-MB-231 breast cancer cells