Latest Research in Ubiquitin and proteasome pathways
13 research papers · 2026 median publication year
Top Research Topics in Ubiquitin and proteasome pathways
- Cancer, Hypoxia, and Metabolism — 3 papers
- Immune cells in cancer — 1 papers
- Ubiquitin and proteasome pathways — 1 papers
- Acute Myeloid Leukemia Research — 1 papers
- Nicotinic Acetylcholine Receptors Study — 1 papers
- Polyamine Metabolism and Applications — 1 papers
- Angiogenesis and VEGF in Cancer — 1 papers
- TGF-β signaling in diseases — 1 papers
- Cancer, Lipids, and Metabolism — 1 papers
- Single-cell and spatial transcriptomics — 1 papers
Highest-Cited Papers
- Tumor Acidosis Boosts Antibody-Driven Cytotoxic Potential of Neutrophils
- Nuclear IDH3A Drives Transcriptional Programs in Melanoma via the YBX1–JUN/FOS Axis
- Reorientation of the SUMOylation landscape and altered L3mbtl2-mediated transcriptional activity in cancer-associated muscle contractile dysfunction
- Tet2 deficiency drives age-related heterogeneous progression of myeloid neoplasms through epigenetic-immune microenvironment imbalance
- The CAGE Framework (Calcium-Gated Excitability): From Single Neurotransmitters to Calcium States in the Tumor Microenvironment
- A hypoxia/necrosis-driven metabolic-epigenetic axis H3K9 lactylation-mediated SKP2 signaling promotes glioblastoma progression
- KDM6A loss enhances oxidative phosphorylation uncovering tissue-level convergent evolution
- Targeting Reactive Species Stress and Nutrient Sensing to Enhance NK-Cell Function: Mechanistic Strategies for Overcoming Pancreatic Cancer Progression and Resistance Through Supplement Therapy
- α-KG conformationally stabilizes nuclear GLUD1-SNAIL complex to fuel trans-endothelial migration in metastasis
- Targeting CKIP ‐1 for Disease Therapy: Cellular Functions, Molecular Networks, and Clinical Potential
- 76 Metabolic Heterogeneity in Brain Tumours: Distinguishing Brain Metastases from Glioblastomas
- Single-cell and spatial transcriptomics reveal glycolytic remodeling and ENO1-associated metabolic vulnerability in hepatoblastoma
- Loss of ST8SIA6 is associated with enhanced MUC16-Integrin β4 signaling and aggressive phenotypes in colorectal cancer