Apelin signaling controls neuroendocrine homeostasis via Atoh8-dependent transcriptional regulation

Apelin is a pleiotropic peptide hormone with established roles in cardiovascular function, angiogenesis, and metabolism, but its contribution to neuroendocrine development and function remains poorly understood. Here, using genetic loss-of-function and inducible gain-of-function approaches in zebrafish, we show that Apelin signaling is dosage-sensitive and required for normal development, reproductive capacity, and physiological homeostasis. Both apelin deficiency and apelin overexpression disrupted embryonic development and reduced adult lifespan, while exhibiting markedly reduced fertility in both sexes. Transcriptomic profiling revealed broad changes in neuroendocrine, hormonal, stress-response, and inflammatory genes with young adult mutant expression profiles resembling those of aged wild-type fish. Both loss and excess of Apelin also disrupted hypothalamic-pituitary-thyroid (HPT) axis development, reducing hypothalamic thyrotropin-releasing hormone-expressing cells, pituitary gonadotropic cell populations, and thyroid gland volume. Transcriptomic analyses identified atoh8, encoding a basic helix-loop-helix transcription factor, as a candidate effector of Apelin signaling. Apelin expression increased atoh8 expression during early embryogenesis, whereas atoh8 mRNA partially rescued developmental defects in apelin mutants, including hypothalamic and pituitary markers, and thyroid volume. Conversely, atoh8 crispants displayed thyroid and pituitary abnormalities, supporting a role for Atoh8 in neuroendocrine development. Together, these findings establish Apelin signaling as a dosage-sensitive regulator of zebrafish development and tissue homeostasis and identify Atoh8 as a functional downstream effector that partially mediates Apelin-dependent HPT axis formation. This work links peptide hormone signaling to neuroendocrine development and provides a framework for investigating its broader contributions to vertebrate physiology, reproduction and aging.

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Publication Details

Journal
bioRxiv (Cold Spring Harbor Laboratory)
Published
2026-11-03
DOI
https://doi.org/10.64898/2026.11.02.763639
Primary Topic
Apelin-related biomedical research
Type
preprint
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preprint

Apelin signaling controls neuroendocrine homeostasis via Atoh8-dependent transcriptional regulation

Caroline Halluin, Romain Madelaine, Anindita Neog, Romain Menard et al.
bioRxiv (Cold Spring Harbor Laboratory)
Apelin-related biomedical research
preprint

Apelin signaling controls neuroendocrine homeostasis via Atoh8-dependent transcriptional regulation

Caroline Halluin, Romain Madelaine, Anindita Neog, Romain Menard, Frederic Bonnet
preprint en

Abstract

Apelin is a pleiotropic peptide hormone with established roles in cardiovascular function, angiogenesis, and metabolism, but its contribution to neuroendocrine development and function remains poorly understood. Here, using genetic loss-of-function and inducible gain-of-function approaches in zebrafish, we show that Apelin signaling is dosage-sensitive and required for normal development, reproductive capacity, and physiological homeostasis. Both apelin deficiency and apelin overexpression disrupted embryonic development and reduced adult lifespan, while exhibiting markedly reduced fertility in both sexes. Transcriptomic profiling revealed broad changes in neuroendocrine, hormonal, stress-response, and inflammatory genes with young adult mutant expression profiles resembling those of aged wild-type fish. Both loss and excess of Apelin also disrupted hypothalamic-pituitary-thyroid (HPT) axis development, reducing hypothalamic thyrotropin-releasing hormone-expressing cells, pituitary gonadotropic cell populations, and thyroid gland volume. Transcriptomic analyses identified atoh8, encoding a basic helix-loop-helix transcription factor, as a candidate effector of Apelin signaling. Apelin expression increased atoh8 expression during early embryogenesis, whereas atoh8 mRNA partially rescued developmental defects in apelin mutants, including hypothalamic and pituitary markers, and thyroid volume. Conversely, atoh8 crispants displayed thyroid and pituitary abnormalities, supporting a role for Atoh8 in neuroendocrine development. Together, these findings establish Apelin signaling as a dosage-sensitive regulator of zebrafish development and tissue homeostasis and identify Atoh8 as a functional downstream effector that partially mediates Apelin-dependent HPT axis formation. This work links peptide hormone signaling to neuroendocrine development and provides a framework for investigating its broader contributions to vertebrate physiology, reproduction and aging.

bioRxiv (Cold Spring Harbor Laboratory)
Mount Desert Island Biological Laboratory (US)
Apelin-related biomedical research
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