Diagnostic yield of cancer predisposition in a nationwide prospective childhood acute leukemia cohort

Abstract The utility of whole-genome sequencing (WGS) for detecting childhood leukemia predisposition remains unclear. We perform a nationwide, prospective, population-based study of 181 children with acute leukemia to assess diagnostic yield and clinical utility of a uniformly applied three-pronged strategy, including systematic phenotyping, germline WGS-based 189-gene panel and tumor sequencing of positive cases. Trio-WGS is performed in 11 high-suspicion families. Nine patients have pathogenic germline alterations: six (3.3%) with leukemia predisposition syndromes ( TP53, CEBPA, DNMT3A , trisomy 21) and three (1.6%) with solid tumor predisposition syndromes ( MSH6, PALB2, SDHA ). Trio-WGS identifies one likely-pathogenic de novo DNMT3A variant. Six of nine diagnoses are previously unrecognized. Findings lead to tailored surveillance in 8/9 patients and treatment modifications in 4/9. Here, we show that this strategy gives a modest diagnostic yield. Nevertheless, the actionability of the findings supports its feasibility and validity in practice. Whether to restrict analysis to leukemia-relevant genes or broaden the panel should reflect local resources and counseling capacity.

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Journal
Nature Communications
Published
2026-09-28
DOI
https://doi.org/10.1038/s41467-026-78170-z
Primary Topic
Acute Myeloid Leukemia Research
Type
article
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Diagnostic yield of cancer predisposition in a nationwide prospective childhood acute leukemia cohort

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Nature Communications
Acute Myeloid Leukemia Research
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Diagnostic yield of cancer predisposition in a nationwide prospective childhood acute leukemia cohort

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article en

Abstract

Abstract The utility of whole-genome sequencing (WGS) for detecting childhood leukemia predisposition remains unclear. We perform a nationwide, prospective, population-based study of 181 children with acute leukemia to assess diagnostic yield and clinical utility of a uniformly applied three-pronged strategy, including systematic phenotyping, germline WGS-based 189-gene panel and tumor sequencing of positive cases. Trio-WGS is performed in 11 high-suspicion families. Nine patients have pathogenic germline alterations: six (3.3%) with leukemia predisposition syndromes ( TP53, CEBPA, DNMT3A , trisomy 21) and three (1.6%) with solid tumor predisposition syndromes ( MSH6, PALB2, SDHA ). Trio-WGS identifies one likely-pathogenic de novo DNMT3A variant. Six of nine diagnoses are previously unrecognized. Findings lead to tailored surveillance in 8/9 patients and treatment modifications in 4/9. Here, we show that this strategy gives a modest diagnostic yield. Nevertheless, the actionability of the findings supports its feasibility and validity in practice. Whether to restrict analysis to leukemia-relevant genes or broaden the panel should reflect local resources and counseling capacity.

Nature CommunicationsVol. 17(1)
Linköping University (SE), Uppsala University (SE), Karolinska University Hospital (SE), Uppsala University Hospital (SE), Lund University (SE), Science for Life Laboratory (SE), Sahlgrenska University Hospital (SE), Karolinska Institutet (SE), Region Västra Götaland (SE), Skåne University Hospital (SE), Linköping University Hospital (SE), Region Östergötland, KTH Royal Institute of Technology (SE), University of Gothenburg (SE), Umeå University (SE)
Openalex Percentile: Top 11%
Acute Myeloid Leukemia Research
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