Follistatin drives AR-independent prostate cancer through BMP signaling inactivation and microenvironmental remodeling

Resistance to androgen receptor (AR)-targeted therapies remains a major clinical challenge in advanced prostate cancer. Here we identify follistatin (FST), TGFβ superfamily antagonist, as a key driver of AR-independence and treatment resistance. During anti-AR therapy, cancer cells progressively lose the positive feedback loop between AR and BMP signaling, leading to FST upregulation that further suppresses this circuit. FST antagonizes BMP11-dependent AR maintenance and upregulates the lineage plasticity regulator SOX9, conferring resistance to enzalutamide while promoting stem-like properties and metastatic capabilities. FST also remodels the tumor microenvironment by driving osteoclastogenesis and compromising T cell effector function. Clinically, FST inversely correlates with PSA levels and associates with poor outcomes in enzalutamide-treated patients. We developed FST-Trap, which effectively neutralizes FST, resensitizes resistant tumors to AR-targeted therapy, and enhances immunotherapy efficacy. This establishes FST as both a biomarker and therapeutic target for AR-independent prostate cancer.

Authors

Institutions

Publication Details

Journal
Proceedings of the National Academy of Sciences
Published
2026-09-24
DOI
https://doi.org/10.1073/pnas.2603564123
Primary Topic
TGF-β signaling in diseases
Type
article
Field-Weighted Citation Impact
0.00
Controls
|||
ALL TIME
JAN
FEB
MAR
APR
MAY
JUN
JUL
AUG
SEP
article

Follistatin drives AR-independent prostate cancer through BMP signaling inactivation and microenvironmental remodeling

Zuyan Fan, Wenjing Su, Liqun Xia, Gonghui Li et al.
Proceedings of the National Academy of Sciences
TGF-β signaling in diseases
article

Follistatin drives AR-independent prostate cancer through BMP signaling inactivation and microenvironmental remodeling

Zuyan Fan, Wenjing Su, Liqun Xia, Gonghui Li, Hongliang Gao, Zhifan Ding, Furong Liu, Fang Dong, Xiaoxian Shi, Wei Xie, Deli Huang, Yizhong Bao, Jing Li, Weijie Zhang, Xu Gao, Biqi Dai, Min Qu, Xinhua Feng, Jun Chen, Chunjie Lin, Rushan Chen, Zhiheng Ji, Yixuan Du, Tianyu Cao, Caiwei Tan
article en

Abstract

Resistance to androgen receptor (AR)-targeted therapies remains a major clinical challenge in advanced prostate cancer. Here we identify follistatin (FST), TGFβ superfamily antagonist, as a key driver of AR-independence and treatment resistance. During anti-AR therapy, cancer cells progressively lose the positive feedback loop between AR and BMP signaling, leading to FST upregulation that further suppresses this circuit. FST antagonizes BMP11-dependent AR maintenance and upregulates the lineage plasticity regulator SOX9, conferring resistance to enzalutamide while promoting stem-like properties and metastatic capabilities. FST also remodels the tumor microenvironment by driving osteoclastogenesis and compromising T cell effector function. Clinically, FST inversely correlates with PSA levels and associates with poor outcomes in enzalutamide-treated patients. We developed FST-Trap, which effectively neutralizes FST, resensitizes resistant tumors to AR-targeted therapy, and enhances immunotherapy efficacy. This establishes FST as both a biomarker and therapeutic target for AR-independent prostate cancer.

Proceedings of the National Academy of SciencesVol. 123(39)
Second Military Medical University (CN), Sir Run Run Shaw Hospital (CN), Zhejiang Cancer Hospital (CN), Changhai Hospital (CN), Zhejiang Hospital (CN), Second Affiliated Hospital of Zhejiang University (CN), Institute of Pharmacology (CZ), First Affiliated Hospital Zhejiang University (CN), Huazhong University of Science and Technology (CN), Zhejiang University (CN)
Openalex Percentile: Top 19%
TGF-β signaling in diseases
AI Navigator

Ask Laika to Summarize, Analyze, and Connect papers live on the map.

Summarize Papers & Methodologies

Extract key findings, datasets, and comparative methods across publications.

Benchmark Rankings & Visual Analytics

Rank top research institutions, authors, funders, topics, and journals by Field-Weighted Citation Impact (FWCI) and paper volume with instant charts.

Connect Distant Disciplines

Bridge topological clusters on the map to find hidden collaborative intersections.