Ceramide synthesis mediates colorectal cancer metastasis through a YAP-driven regenerative program

Mechanisms by which primary tumor cells acquire metastatic capability through metabolic and signaling adaptations are currently poorly understood. We demonstrate that tumor-intrinsic ceramide metabolism, amplified by dietary fat, initiates colorectal cancer metastasis. We observed that dietary fat exposure triggers a sustained increase in de novo ceramide biosynthesis, mediated by the dihydroceramide desaturase Degs1 . Ceramide accumulation activates yes-associated protein (YAP) through protein phosphatase 2A (PP2A)–mediated dephosphorylation, promoting a durable shift toward a distinct YAP-driven regenerative (YAP-DR) program, marked by Basp1 , that promotes metastasis. Selective elimination of Basp1 high cancer cells prevented metastatic seeding. Degs1 loss reduced ceramide levels, YAP activity, YAP-DR signatures, and metastasis without affecting primary tumor growth, whereas blocking ceramide degradation enhanced YAP activity and metastasis. These findings identify ceramide-induced YAP signaling as a key mediator of metastatic initiation, operating independently of primary tumor expansion.

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Journal
Science
Published
2026-09-24
DOI
https://doi.org/10.1126/science.adw8520
Primary Topic
Sphingolipid Metabolism and Signaling
Type
article
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0.00
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article

Ceramide synthesis mediates colorectal cancer metastasis through a YAP-driven regenerative program

Maria Fedorova, Autumn Gabrielle York, Matthew G. Vander Heiden, Jonas Rösler et al.
Science
Sphingolipid Metabolism and Signaling
article

Ceramide synthesis mediates colorectal cancer metastasis through a YAP-driven regenerative program

Maria Fedorova, Autumn Gabrielle York, Matthew G. Vander Heiden, Jonas Rösler, Besim Öğretmen, Albert Sickmann, Sven Heiles, Akhouri Kishore Raghawan, Charles A. Whittaker, Ömer H. Yilmaz, Nilay S. Sethi, Odai Darawshi, Vikram Deshpande, José Antonio Ortiz, Oliver Johannes Schmitz, Alpaslan Tasdogan, Edrees H. Rashan, George Eng, Abdullah Burak Yıldız, Feyza Cansiz, Manon Bulliard, Sven W. Meckelmann, Swagata Goswami, Joseph C. Sedlak, Chiara Alquati, Gabriele Allies, Upasana Das Adhikari, Benjamin J. Read, Ulf P. Neumann, Kevin J. Williams, Douglas S. Kwon, Felix-Levin Hormann, Qiming Zhang, Zhixin Li, Osman H. Yilmaz, Wesley W Grace, Cigdem Elif Celik, Sabhyata Sedhain, Constantin P. Krempe, Anup Jnawali, Ethan Reich, Chiashin Chi, Chesta Jain
article en

Abstract

Mechanisms by which primary tumor cells acquire metastatic capability through metabolic and signaling adaptations are currently poorly understood. We demonstrate that tumor-intrinsic ceramide metabolism, amplified by dietary fat, initiates colorectal cancer metastasis. We observed that dietary fat exposure triggers a sustained increase in de novo ceramide biosynthesis, mediated by the dihydroceramide desaturase Degs1 . Ceramide accumulation activates yes-associated protein (YAP) through protein phosphatase 2A (PP2A)–mediated dephosphorylation, promoting a durable shift toward a distinct YAP-driven regenerative (YAP-DR) program, marked by Basp1 , that promotes metastasis. Selective elimination of Basp1 high cancer cells prevented metastatic seeding. Degs1 loss reduced ceramide levels, YAP activity, YAP-DR signatures, and metastasis without affecting primary tumor growth, whereas blocking ceramide degradation enhanced YAP activity and metastasis. These findings identify ceramide-induced YAP signaling as a key mediator of metastatic initiation, operating independently of primary tumor expansion.

ScienceVol. 393(6818)
Broad Institute (US), Albert Einstein College of Medicine (US), Beth Israel Deaconess Medical Center (US), Harvard University (US), Medical University of South Carolina (US), University of California, Los Angeles (US), University of Washington (US), Ragon Institute of MGH, MIT and Harvard (US), Maastricht University (NL), Ege University (TR), Massachusetts General Hospital (US), MUSC Hollings Cancer Center (US), Hacettepe University Hospital (TR), Dana-Farber Cancer Institute (US), Essen University Hospital (DE), Leibniz Institute for Analytical Sciences - ISAS (DE), Gilead Sciences (Spain) (ES), Gilead Sciences (United States) (US), University Hospital Carl Gustav Carus (DE), University of Duisburg-Essen (DE), Massachusetts Institute of Technology (US), Hacettepe University (TR), Technische Universität Dresden (DE), Ruhr University Bochum (DE), University of Bologna (IT), University of California, Berkeley (US)
Openalex Percentile: Top 19%
Sphingolipid Metabolism and Signaling
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