The liver niche accelerates, while the lung niche restrains secondary spread of breast cancer

Abstract Breast cancer progression to visceral organs such as the lung and liver is associated with poor prognosis. Yet, these organs have diverse cellular microenvironments that may differentially influence cancer cell behavior. Here, we show that breast cancer metastases differ in their capacity for secondary seeding: the liver fosters metastasis-promoting properties and boosts secondary spread, whereas the lung restricts these processes, favoring local growth. Using patient data and rapid autopsy tissues, mouse models with barcode-mediated tracing, niche-labeling technology, and single-cell analysis of tumor cells and their direct microenvironment, we dissect the cellular and molecular microenvironmental factors underlying this differential behavior. Mechanistically, we identify BMP2-producing endothelial cells as critical players within the liver metastatic niche, enhancing metastasis-to-metastasis dissemination. Targeting the BMP2 signaling axis suppresses secondary spread of liver-derived breast cancer lesions. Altogether, we reveal site-specific behavior of lung and liver metastases in breast cancer, highlighting microenvironmental mechanisms and therapeutic opportunities for intervention.

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Publication Details

Journal
Cancer Discovery
Published
2026-09-22
DOI
https://doi.org/10.1158/2159-8290.cd-25-1652
Primary Topic
Cancer Cells and Metastasis
Type
article
Field-Weighted Citation Impact
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article

The liver niche accelerates, while the lung niche restrains secondary spread of breast cancer

Fabienne Dominique Schwab, Marcus Vetter, Andreas N. WICKI, Lauren L. Ozimski et al.
Cancer Discovery
Cancer Cells and Metastasis
article

The liver niche accelerates, while the lung niche restrains secondary spread of breast cancer

Fabienne Dominique Schwab, Marcus Vetter, Andreas N. WICKI, Lauren L. Ozimski, Gaël Auray, Heike Frauchiger-Heuer, Giada Pontecorvi, Zacharias Kontarakis, Alexander Ring, Jianwen Zhou, Magdalena K. Sznurkowska, David Gremmelspacher, Karin Strittmatter, Kirsten D. Mertz, Simran Asawa, Yu Wei Zhang, Verdon Taylor, Ilaria Malanchi, Werner Josef Kovacs, Francesc Castro-Giner, Matteo Ligorio, Selina Budinjas, Francesca Albrecht, Massimo Saini, Ilona Krol, Nicola Aceto, Ana Gvozdenovic, Claudio Giachino, Marko Vujanovic, Yongzhan Zhang, Irene D'Anna
article en

Abstract

Abstract Breast cancer progression to visceral organs such as the lung and liver is associated with poor prognosis. Yet, these organs have diverse cellular microenvironments that may differentially influence cancer cell behavior. Here, we show that breast cancer metastases differ in their capacity for secondary seeding: the liver fosters metastasis-promoting properties and boosts secondary spread, whereas the lung restricts these processes, favoring local growth. Using patient data and rapid autopsy tissues, mouse models with barcode-mediated tracing, niche-labeling technology, and single-cell analysis of tumor cells and their direct microenvironment, we dissect the cellular and molecular microenvironmental factors underlying this differential behavior. Mechanistically, we identify BMP2-producing endothelial cells as critical players within the liver metastatic niche, enhancing metastasis-to-metastasis dissemination. Targeting the BMP2 signaling axis suppresses secondary spread of liver-derived breast cancer lesions. Altogether, we reveal site-specific behavior of lung and liver metastases in breast cancer, highlighting microenvironmental mechanisms and therapeutic opportunities for intervention.

Cancer Discovery
University of Basel (CH), University of Zurich (CH), The Francis Crick Institute (GB), University Hospital of Basel (CH), ETH Zurich (CH), Kantonsspital Baselland (CH), University Hospital of Zurich (CH), Department of Biomedicine Basel (CH), The University of Texas Southwestern Medical Center (US)
Good health and well-being
Openalex Percentile: Top 14%
Cancer Cells and Metastasis
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