Restoring Cerebral Venous Outflow: Right Internal Jugular to Subclavian Vein Bypass After Ligation in Thoracic Outlet Syndrome

BackgroundIntracranial venous hypertension (IVH) is an underrecognised cause of persistent craniofacial symptoms, often presenting with nonspecific features such as pressure-type headaches, pulsatile tinnitus, and visual disturbances. This case illustrates a complex diagnostic dilemma in which symptoms classically attributed to Eagle Syndrome masked another vascular cause of intracranial venous hypertension. The patient provided informed consent for treatment and for the use of his clinical information in this case report.Case SummaryA 59-year-old woman presented with refractory craniofacial symptoms initially attributed to bilateral Eagle Syndrome. Despite bilateral stylodectomy, internal jugular vein (IJV) decompression, C1 transverse process resection, and right occipital sinus stenting, her symptoms persisted. During right-sided intervention for Eagle Syndrome, the right IJV was found to have been previously ligated at the clavicle during venous thoracic outlet syndrome surgery. Venous manometry demonstrated elevated pressures within the right IJV (32 mmHg) and torcula (29 mmHg), supporting IVH secondary to impaired venous outflow. Definitive treatment consisted of a right IJV-subclavian vein (SCV) bypass using a triple-panel great saphenous vein (GSV) graft to achieve an adequate conduit diameter. The postoperative course was complicated by early graft thrombosis and neck hematoma, requiring urgent hybrid open and endovascular reintervention. This included suction thrombectomy, angioplasty, sequential stenting of the bypass using bare-metal, covered, and dedicated venous stents, and open repair of a graft tear with bovine pericardial patch angioplasty. Following reintervention, the bypass remained patent on follow-up imaging, with complete resolution of headaches, pulsatile tinnitus, and visual symptoms.ConclusionThis case highlights the importance of comprehensive venous evaluation in patients with persistent craniofacial symptoms, particularly following thoracic outlet interventions. It also demonstrates the feasibility of triple-panel GSV grafting for central venous reconstruction and the value of a hybrid surgical-endovascular approach in managing complex venous complications.

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Journal
Vascular and Endovascular Surgery
Published
2026-09-21
DOI
https://doi.org/10.1177/15385744261491474
Primary Topic
Oropharyngeal Anatomy and Pathologies
Type
article
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article

Restoring Cerebral Venous Outflow: Right Internal Jugular to Subclavian Vein Bypass After Ligation in Thoracic Outlet Syndrome

Luca Borruso, Dushan Miladinovic, Eshan Affan, Steven Dubenec
Vascular and Endovascular Surgery
Oropharyngeal Anatomy and Pathologies
article

Restoring Cerebral Venous Outflow: Right Internal Jugular to Subclavian Vein Bypass After Ligation in Thoracic Outlet Syndrome

Luca Borruso, Dushan Miladinovic, Eshan Affan, Steven Dubenec
article en

Abstract

BackgroundIntracranial venous hypertension (IVH) is an underrecognised cause of persistent craniofacial symptoms, often presenting with nonspecific features such as pressure-type headaches, pulsatile tinnitus, and visual disturbances. This case illustrates a complex diagnostic dilemma in which symptoms classically attributed to Eagle Syndrome masked another vascular cause of intracranial venous hypertension. The patient provided informed consent for treatment and for the use of his clinical information in this case report.Case SummaryA 59-year-old woman presented with refractory craniofacial symptoms initially attributed to bilateral Eagle Syndrome. Despite bilateral stylodectomy, internal jugular vein (IJV) decompression, C1 transverse process resection, and right occipital sinus stenting, her symptoms persisted. During right-sided intervention for Eagle Syndrome, the right IJV was found to have been previously ligated at the clavicle during venous thoracic outlet syndrome surgery. Venous manometry demonstrated elevated pressures within the right IJV (32 mmHg) and torcula (29 mmHg), supporting IVH secondary to impaired venous outflow. Definitive treatment consisted of a right IJV-subclavian vein (SCV) bypass using a triple-panel great saphenous vein (GSV) graft to achieve an adequate conduit diameter. The postoperative course was complicated by early graft thrombosis and neck hematoma, requiring urgent hybrid open and endovascular reintervention. This included suction thrombectomy, angioplasty, sequential stenting of the bypass using bare-metal, covered, and dedicated venous stents, and open repair of a graft tear with bovine pericardial patch angioplasty. Following reintervention, the bypass remained patent on follow-up imaging, with complete resolution of headaches, pulsatile tinnitus, and visual symptoms.ConclusionThis case highlights the importance of comprehensive venous evaluation in patients with persistent craniofacial symptoms, particularly following thoracic outlet interventions. It also demonstrates the feasibility of triple-panel GSV grafting for central venous reconstruction and the value of a hybrid surgical-endovascular approach in managing complex venous complications.

Vascular and Endovascular Surgery
Royal Prince Alfred Hospital (AU), Concord Repatriation General Hospital (AU)
Good health and well-being
Openalex Percentile: Top 9%
Oropharyngeal Anatomy and Pathologies
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