SLC7A6OS Founder Mutation: A Rare Cause of Progressive Myoclonus Epilepsy Dated to 1100 Years Ago

BACKGROUND: SLC7A6OS c.191A>G is a rare, autosomal recessive cause of progressive myoclonus epilepsy (PME). The c.191A>G variant, first discovered in two families from Türkiye and Portugal, was recently identified in three additional probands from the USA, all of Puerto Rican ancestry. OBJECTIVES: We sought to refine the SLC7A6OS-PME phenotype and determine whether all families inherited the variant from the same common ancestor. METHODS: Clinical and genotyping data were obtained from all five families. Haplotype analysis using single nucleotide polymorphism arrays to investigate a possible common ancestor was performed. RESULTS: Shared haplotypes suggest all five families inherited the SLC7A6OS variant from a common ancestor approximately 1100 years ago. Subsequent dating estimates were consistent with migration patterns between the eastern Mediterranean, Iberia, and Puerto Rico. CONCLUSIONS: Our findings strengthen the previous evidence for SLC7A6OS as a cause of PME and highlight a founder effect in regions with migratory links to Iberia. © 2026 The Author(s). Movement Disorders published by Wiley Periodicals LLC on behalf of International Parkinson and Movement Disorder Society.

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Publication Details

Journal
Movement Disorders
Published
2026-09-18
DOI
https://doi.org/10.1002/mds.70516
Primary Topic
Glycogen Storage Diseases and Myoclonus
Type
article
Field-Weighted Citation Impact
0.00

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article

SLC7A6OS Founder Mutation: A Rare Cause of Progressive Myoclonus Epilepsy Dated to 1100 Years Ago

Krystal Sully, Mered Parnes, Karen Oliver, Nerses Bebek et al.
Movement Disorders
Glycogen Storage Diseases and Myoclonus
article

SLC7A6OS Founder Mutation: A Rare Cause of Progressive Myoclonus Epilepsy Dated to 1100 Years Ago

Krystal Sully, Mered Parnes, Karen Oliver, Nerses Bebek, Bronwyn E. Grinton, Colin A. Ellis, Anna‐Elina Lehesjoki, Sara Cabet, Volkan Taşdemir, Gaëtan Lesca, Laure Mazzola, Samuel F. Berkovic, Melanie Bahlo, Mariam Hull, Jacob E. Munro, Laina Lusk, Betül Baykan, Pamela Pojomovsky McDonnell
article en

Abstract

BACKGROUND: SLC7A6OS c.191A>G is a rare, autosomal recessive cause of progressive myoclonus epilepsy (PME). The c.191A>G variant, first discovered in two families from Türkiye and Portugal, was recently identified in three additional probands from the USA, all of Puerto Rican ancestry. OBJECTIVES: We sought to refine the SLC7A6OS-PME phenotype and determine whether all families inherited the variant from the same common ancestor. METHODS: Clinical and genotyping data were obtained from all five families. Haplotype analysis using single nucleotide polymorphism arrays to investigate a possible common ancestor was performed. RESULTS: Shared haplotypes suggest all five families inherited the SLC7A6OS variant from a common ancestor approximately 1100 years ago. Subsequent dating estimates were consistent with migration patterns between the eastern Mediterranean, Iberia, and Puerto Rico. CONCLUSIONS: Our findings strengthen the previous evidence for SLC7A6OS as a cause of PME and highlight a founder effect in regions with migratory links to Iberia. © 2026 The Author(s). Movement Disorders published by Wiley Periodicals LLC on behalf of International Parkinson and Movement Disorder Society.

Movement Disorders
Université Claude Bernard Lyon 1 (FR), Centre National de la Recherche Scientifique (FR), Children's Hospital of Philadelphia (US), University of Helsinki (FI), Inserm (FR), The University of Melbourne (AU), Lyon College (US), Walter and Eliza Hall Institute of Medical Research (AU), Centre de Recherche en Neurosciences de Lyon (FR), Hospices Civils de Lyon (FR), Institut NeuroMyoGène (FR), Texas Children's Hospital (US), Centre Hospitalier Universitaire de Saint-Étienne (FR), Austin Health (AU), Folkhälsans Forskningscentrum (FI), Istanbul University (TR), University of Pennsylvania (US)
National Health and Medical Research Council, National Institute of Neurological Disorders and Stroke
Reduced inequalities
Openalex Percentile: Top 10%
Glycogen Storage Diseases and Myoclonus
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