Balancing lipid synthesis and oxidation promotes B cell response to vaccination during immunosuppression

Pharmacologic immunosuppression is essential for preventing organ rejection and controlling autoimmunity, but profoundly impairs humoral immunity, increasing the risk of vaccine failure and infection. The mechanisms by which immunosuppressive therapies disrupt human B cell responses remain poorly defined. Here, we identified dysregulated lipid metabolism as a central determinant of impaired vaccine response in solid organ transplant recipients (SOTRs). Using high-dimensional immune profiling, single-cell transcriptomics, and functional metabolic assays, we found that effective B cell responses required a homeostatic balance between lipid synthesis and fatty acid oxidation. The widely used immunosuppressive agent, mycophenolic acid (MPA) was strongly associated with vaccine non-response and induced excessive lipid synthesis, lipid accumulation, and mitochondrial stress in B cells. In contrast, CD11c+ B cells retained the capacity to differentiate into plasmablasts in the presence of MPA through elevated expression of CPT1A, a mitochondrial fatty acid transporter, and enhanced fatty acid oxidation. These cells were found to be a key feature of early effective vaccine responses in healthy individuals and SOTRs. Notably, pharmacologic inhibition of cholesterol synthesis partially restored plasmablast differentiation in the presence of MPA. These findings identify B cell lipid metabolism as a critical and targetable regulator of human humoral immunity during immunosuppression.

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Publication Details

Journal
Journal of Clinical Investigation
Published
2026-09-17
DOI
https://doi.org/10.1172/jci205170
Primary Topic
Renal Transplantation Outcomes and Treatments
Type
article
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article

Balancing lipid synthesis and oxidation promotes B cell response to vaccination during immunosuppression

Justin R. Bailey, Luca Biavati, Mark A. Robien, Samuel B. Warner et al.
Journal of Clinical Investigation
Renal Transplantation Outcomes and Treatments
article

Balancing lipid synthesis and oxidation promotes B cell response to vaccination during immunosuppression

Justin R. Bailey, Luca Biavati, Mark A. Robien, Samuel B. Warner, Hongkai Ji, Nicole Skinner, Santosh Dhakal, Dorry L. Segev, Elizabeth A. Thompson, Sabra L. Klein, Katerina Roznik, Laila Stoddart, William A. Werbel, Alexis Figueroa, Erika L. Pearce, Christian P. Larsen, Andrew H. Karaba, Robert D. Leone, Edward J. Pearce, Peter S. Heeger, Andrea L. Cox, Nadine Rouphael, Aaron A. R. Tobian, Joel N. Blankson, Yolanda Eby, Shuai Li, Karli Redinger, Laura A. Sena
article en

Abstract

Pharmacologic immunosuppression is essential for preventing organ rejection and controlling autoimmunity, but profoundly impairs humoral immunity, increasing the risk of vaccine failure and infection. The mechanisms by which immunosuppressive therapies disrupt human B cell responses remain poorly defined. Here, we identified dysregulated lipid metabolism as a central determinant of impaired vaccine response in solid organ transplant recipients (SOTRs). Using high-dimensional immune profiling, single-cell transcriptomics, and functional metabolic assays, we found that effective B cell responses required a homeostatic balance between lipid synthesis and fatty acid oxidation. The widely used immunosuppressive agent, mycophenolic acid (MPA) was strongly associated with vaccine non-response and induced excessive lipid synthesis, lipid accumulation, and mitochondrial stress in B cells. In contrast, CD11c+ B cells retained the capacity to differentiate into plasmablasts in the presence of MPA through elevated expression of CPT1A, a mitochondrial fatty acid transporter, and enhanced fatty acid oxidation. These cells were found to be a key feature of early effective vaccine responses in healthy individuals and SOTRs. Notably, pharmacologic inhibition of cholesterol synthesis partially restored plasmablast differentiation in the presence of MPA. These findings identify B cell lipid metabolism as a critical and targetable regulator of human humoral immunity during immunosuppression.

Journal of Clinical Investigation
Cedars-Sinai Medical Center (US), Johns Hopkins University (US), Emory University (US), Johns Hopkins Medicine (US), The Ohio State University Wexner Medical Center (US), HOPE Clinic (US), National Institute of Allergy and Infectious Diseases (US)
Good health and well-being
Openalex Percentile: Top 8%
Renal Transplantation Outcomes and Treatments
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