Knockdown of VNN1 protects cardiac myoblasts from hypoxia/reoxygenation injury via the PI3K/Akt pathway

activating the PI3K/Akt pathway, highlighting VNN1 as a potential target for myocardial I/R injury.

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Publication Details

Journal
Korean Journal of Physiology and Pharmacology
Published
2026-09-16
DOI
https://doi.org/10.4196/kjpp.26.034
Primary Topic
Nitric Oxide and Endothelin Effects
Type
article
Field-Weighted Citation Impact
0.00
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article

Knockdown of VNN1 protects cardiac myoblasts from hypoxia/reoxygenation injury via the PI3K/Akt pathway

Yu Fang, XuJiao Hu
Korean Journal of Physiology and Pharmacology
Nitric Oxide and Endothelin Effects
article

Knockdown of VNN1 protects cardiac myoblasts from hypoxia/reoxygenation injury via the PI3K/Akt pathway

Yu Fang, XuJiao Hu
article en

Abstract

No abstract available for this paper.

Korean Journal of Physiology and Pharmacology
Ningbo University Affiliated Hospital (CN)
Good health and well-being
Openalex Percentile: Top 12%
Nitric Oxide and Endothelin Effects
DOIOpen Access PDFOpenAlex
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Knockdown of VNN1 protects cardiac myoblasts from hypoxia/reoxygenation injury via the PI3K/Akt pathway — Yu Fang, XuJiao Hu · Korean Journal of Physiology and Pharmacology (2026) | TGRS Research Map | TGRS