Intestinal infections establish antigen-specific, long-lived memory CD4+ T cells in the brain and meninges

Abstract The meninges form the border between the brain and periphery and house a rich network of immune cells. Here we show that gastrointestinal challenges (intracellular or extracellular bacteria and parasites) reshape the nature of CD4 + T cells in the dura mater, the outer meningeal layer, changing the dominant polarization states to T helper (T H ) 1, T H 17 and T H 2 cells, respectively, with differing cytokine profiles. This occurs via CXCR6–CXCL16-dependent migration of gut-activated CD4 + T cells to the central nervous system, where they establish long-lived memory populations around the dural venous sinuses, within dural lymphoid aggregates and in the brain. Functionally, these orally primed dural CD4 + T were capable of rapid, antigen-specific recall responses, proliferating and producing cytokines upon intravenous rechallenge. Our findings reveal a direct link between intestinal and dural immunity, enabling the central nervous system borders to acquire immunological memory of gut microorganisms, a major source of bloodborne pathogens capable of reaching the brain via fenestrated dural vasculature.

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Publication Details

Journal
Nature Neuroscience
Published
2026-09-08
DOI
https://doi.org/10.1038/s41593-026-02428-4
Primary Topic
Bacterial Infections and Vaccines
Type
article
Field-Weighted Citation Impact
0.00

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article

Intestinal infections establish antigen-specific, long-lived memory CD4+ T cells in the brain and meninges

Andrew P. Stewart, Zewen Kelvin Tuong, Katherine Harcourt, Nathan Richoz et al.
Nature Neuroscience
Bacterial Infections and Vaccines
article

Intestinal infections establish antigen-specific, long-lived memory CD4+ T cells in the brain and meninges

Andrew P. Stewart, Zewen Kelvin Tuong, Katherine Harcourt, Nathan Richoz, David R. Withers, Rafael Di Marco Barros, Mia Cabantous, Colin Y.C. Lee, Gad Frankel, Ana Peñalver, Menna R. Clatworthy, Anaïs Portet, Eleanor Gillman, Tetsuo Hasegawa, Aaron Fleming, Tammie Tao Min Sow, David Ruano-Gallego, Karen Neish, David A. Posner, Miles Bremridge, Simon Clare
article en

Abstract

Abstract The meninges form the border between the brain and periphery and house a rich network of immune cells. Here we show that gastrointestinal challenges (intracellular or extracellular bacteria and parasites) reshape the nature of CD4 + T cells in the dura mater, the outer meningeal layer, changing the dominant polarization states to T helper (T H ) 1, T H 17 and T H 2 cells, respectively, with differing cytokine profiles. This occurs via CXCR6–CXCL16-dependent migration of gut-activated CD4 + T cells to the central nervous system, where they establish long-lived memory populations around the dural venous sinuses, within dural lymphoid aggregates and in the brain. Functionally, these orally primed dural CD4 + T were capable of rapid, antigen-specific recall responses, proliferating and producing cytokines upon intravenous rechallenge. Our findings reveal a direct link between intestinal and dural immunity, enabling the central nervous system borders to acquire immunological memory of gut microorganisms, a major source of bloodborne pathogens capable of reaching the brain via fenestrated dural vasculature.

Nature Neuroscience
MRC Laboratory of Molecular Biology (GB), University of Cambridge (GB), Wellcome Sanger Institute (GB), University of Oxford (GB), Centro de Biología Molecular Severo Ochoa (ES), MRC Epidemiology Unit (GB), Molecular Immunity Unit (GB), Imperial College London (GB)
Wellcome Trust, Medical Research Council
Openalex Percentile: Top 13%
Bacterial Infections and Vaccines
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