Inflammatory bone marrow microenvironment impairs the therapeutic effect of daratumumab-lenalidomide in multiple myeloma

Daratumumab-lenalidomide (DR) combination therapy improves multiple myeloma (MM) outcomes, yet some patients respond poorly. We examined 21 DR pre-treatment samples from the IFM2017-03 phase 3 trial using single-cell and bulk multiomics approaches to identify determinants of response. Analysis of the bone marrow environment revealed a coordinated inflammatory state across immune and myeloma cells associated with DR resistance. We identified upregulated NF-κB signaling genes in monocytes of non-responders, alongside increased proinflammatory ISG + T cells and attenuated NK cell CD16 expression, suggesting an impairment of antibody-dependent cellular cytotoxicity essential for daratumumab efficacy. Concurrently, myeloma cells displayed enhanced NF-κB activation, supporting convergent tumor and immune inflammatory programs. We further developed an eight-gene NF-κB-related signature that robustly predicted treatment response in independent bone marrow (n = 28, AUC = 0.81) and peripheral blood (n = 40, AUC = 0.77) samples. Our study underscores an NF-κB-associated inflammatory axis underlying poor DR response and suggests that targeting inflammatory pathways a potential therapeutic avenue warranting further investigation.

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Publication Details

Journal
Biomarker Research
Published
2026-09-05
DOI
https://doi.org/10.1186/s40364-026-00992-2
Primary Topic
Multiple Myeloma Research and Treatments
Type
article
Field-Weighted Citation Impact
0.00

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article

Inflammatory bone marrow microenvironment impairs the therapeutic effect of daratumumab-lenalidomide in multiple myeloma

Malo Leprohon, Aïcha Ouelkdite, Gauthier Decool, Benjamin Podvin et al.
Biomarker Research
Multiple Myeloma Research and Treatments
article

Inflammatory bone marrow microenvironment impairs the therapeutic effect of daratumumab-lenalidomide in multiple myeloma

Malo Leprohon, Aïcha Ouelkdite, Gauthier Decool, Benjamin Podvin, Silvia Gaggero, Ludovic Martinet, Jérôme Moreaux, Jill Corre, Sophie Groux‐Degroote, Guillaume Escure, Rémi Tilmont, Wenfei Jin, Angélina Kasprowicz, Céline Villenet, Lama Hasan Bou Issa, Adeline Cozzani, Yanick Njosse Tchantchou, Emilie Wauquier, Léa Fléchon, Thierry Facon, Wenwen Cheng, Xavier Leleu, Bertrand Arnulf, Suman Mitra, Noémie Carlier, Martin Figeac, Mojtaba Shekarkar, Salomon Manier
article en

Abstract

Daratumumab-lenalidomide (DR) combination therapy improves multiple myeloma (MM) outcomes, yet some patients respond poorly. We examined 21 DR pre-treatment samples from the IFM2017-03 phase 3 trial using single-cell and bulk multiomics approaches to identify determinants of response. Analysis of the bone marrow environment revealed a coordinated inflammatory state across immune and myeloma cells associated with DR resistance. We identified upregulated NF-κB signaling genes in monocytes of non-responders, alongside increased proinflammatory ISG + T cells and attenuated NK cell CD16 expression, suggesting an impairment of antibody-dependent cellular cytotoxicity essential for daratumumab efficacy. Concurrently, myeloma cells displayed enhanced NF-κB activation, supporting convergent tumor and immune inflammatory programs. We further developed an eight-gene NF-κB-related signature that robustly predicted treatment response in independent bone marrow (n = 28, AUC = 0.81) and peripheral blood (n = 40, AUC = 0.77) samples. Our study underscores an NF-κB-associated inflammatory axis underlying poor DR response and suggests that targeting inflammatory pathways a potential therapeutic avenue warranting further investigation.

Biomarker ResearchVol. 14(1)
Centre National de la Recherche Scientifique (FR), Inserm (FR), Institut Universitaire de France (FR), Université de Lille (FR), Centre Hospitalier Universitaire de Lille (FR), Southern University of Science and Technology (CN), Centre de Recherches en Cancérologie de Toulouse (FR), Lille’s Cardiology Hospital (FR), Shanghai Institute of Nutrition and Health (CN), Centre Hospitalier Universitaire de Poitiers (FR), Unité de Glycobiologie Structurale et Fonctionnelle (FR), Centre de Recherche en Cancérologie de Lyon (FR), Hôpital Saint-Louis (FR), Institut de Génétique Humaine (FR), Institut de Biologie de Lille (FR), University of Chinese Academy of Sciences (CN)
Johnson and Johnson
No poverty
Openalex Percentile: Top 10%
Multiple Myeloma Research and Treatments
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