SET7-mediated methylation of IRF3 at lysine 98 attenuates antiviral innate immunity

SET7 belongs to the SET domain (SETD) methyltransferase family, which catalyzes the monomethylation of lysine residues in histones and nonhistone proteins. This process can either enhance or suppress gene activation. Interferon regulatory factor 3 (IRF3) is a key transcription factor in the type I interferon (IFN) signaling pathway. This pathway is controlled by multiple posttranslational modifications that finely tune IRF3’s function. Here, we identify SET7 as a negative regulator of IRF3. SET7 interacts with IRF3 and catalyzes the monomethylation of IRF3 at lysine 98. This modification decreases IRF3 phosphorylation, dimerization, and subsequent nuclear translocation, thereby inhibiting the production of downstream type I interferons. Furthermore, zebrafish lacking set7 , as well as those treated with the inhibitor (R)-PFI-2, exhibits greater resistance to viral infection. Set7 -deficient mice also exhibit greater resistance to RNA and DNA viral infections. Our findings reveal a role for SET7 in regulating antiviral innate immunity and provide insight into the IRF3 monomethylation that affects its activation.

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Publication Details

Journal
Proceedings of the National Academy of Sciences
Published
2026-08-25
DOI
https://doi.org/10.1073/pnas.2601297123
Primary Topic
interferon and immune responses
Type
article
Field-Weighted Citation Impact
0.00

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article

SET7-mediated methylation of IRF3 at lysine 98 attenuates antiviral innate immunity

Chunchun Zhu, Shuke Jia, Wenhua Li, Wuhan Xiao et al.
Proceedings of the National Academy of Sciences
interferon and immune responses
article

SET7-mediated methylation of IRF3 at lysine 98 attenuates antiviral innate immunity

Chunchun Zhu, Shuke Jia, Wenhua Li, Wuhan Xiao, Huangyuan Zha, Xueyi Sun, Xing Liu, Zixuan Wang, Jinhua Tang, Hongyan Deng, Yiman Luo, Yuhan Xiang, Xiaoyun Chen, Wen Liu, Jiale Hua
article en

Abstract

SET7 belongs to the SET domain (SETD) methyltransferase family, which catalyzes the monomethylation of lysine residues in histones and nonhistone proteins. This process can either enhance or suppress gene activation. Interferon regulatory factor 3 (IRF3) is a key transcription factor in the type I interferon (IFN) signaling pathway. This pathway is controlled by multiple posttranslational modifications that finely tune IRF3’s function. Here, we identify SET7 as a negative regulator of IRF3. SET7 interacts with IRF3 and catalyzes the monomethylation of IRF3 at lysine 98. This modification decreases IRF3 phosphorylation, dimerization, and subsequent nuclear translocation, thereby inhibiting the production of downstream type I interferons. Furthermore, zebrafish lacking set7 , as well as those treated with the inhibitor (R)-PFI-2, exhibits greater resistance to viral infection. Set7 -deficient mice also exhibit greater resistance to RNA and DNA viral infections. Our findings reveal a role for SET7 in regulating antiviral innate immunity and provide insight into the IRF3 monomethylation that affects its activation.

Proceedings of the National Academy of SciencesVol. 123(35)
Chinese Academy of Sciences (CN), Wuhan University (CN), Qingdao National Laboratory for Marine Science and Technology (CN), Institute of Hydrobiology, Biology Centre, Academy of Sciences of the Czech Republic (CZ), University of Chinese Academy of Sciences (CN)
National Natural Science Foundation of China
Openalex Percentile: Top 17%
interferon and immune responses
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