DEEP SYSTEMISING AS EVOLUTIONARILY RETAINED COGNITIVE ARCHITECTURE: Causal-Depth Processing as a Qualitatively Distinct Architecture in High-Systemising Autistic Profiles Across Neurological, Evolutionary, Psychological, and Organisational Levels of

This paper proposes a cross-level theoretical account of high-systemising autistic cognition. The central claim is that autistic systemising does not simply occur more frequently than neurotypical systemising — it terminates at a different level. Neurotypical systemising stops at the surface pattern sufficient for social coordination; autistic causal-depth systemising continues to the underlying causal logic that makes the pattern necessary. That distinction — a difference of kind rather than degree — is the framework's central theoretical proposition. The argument develops across four levels of analysis: Neurological: reduced thalamic gating efficiency, arising from excitatory-inhibitory imbalance and aberrant precision weighting, produces a system in which prior beliefs are insufficiently weighted to halt processing at the template level. Evolutionary: specific Neanderthal-derived variants enriched in autistic individuals (Pauly et al., 2024), interpreted as consistent with a retained ancestral processing style maintained through de novo regeneration and episodic frequency-dependent selective value. Psychological: causal-depth processing generates characteristic costs (generative masking, rest-resistant burnout, HPA axis amplification) and outputs (first-principles problem-solving, structural identification of system failure). Sociological: the architecture is proposed to function as a structural catalyst at institutional change points, at characteristic personal cost. A processing-budget construct is developed to explain within-population variation in observable systemising, identifying a clinically significant, budget-depleted subpopulation that is indistinguishable from architectural non-systemisers on current instruments. Nine testable hypotheses (H1–H9) are generated, together with a four-phase empirical research programme gated on the development of a validated processing-depth instrument. The paper is a theoretical synthesis rather than an empirical study. All citations have been verified against primary sources; inferential extensions beyond the cited evidence are flagged explicitly throughout. This is a pre-print made available for scholarly engagement prior to peer review.

Authors

Publication Details

Journal
Zenodo (CERN European Organization for Nuclear Research)
Published
2026-07-01
DOI
https://doi.org/10.5281/zenodo.21093658
Primary Topic
Autism Spectrum Disorder Research
Type
preprint
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DEEP SYSTEMISING AS EVOLUTIONARILY RETAINED COGNITIVE ARCHITECTURE: Causal-Depth Processing as a Qualitatively Distinct Architecture in High-Systemising Autistic Profiles Across Neurological, Evolutionary, Psychological, and Organisational Levels of

Matthew Richard Hodgson
Zenodo (CERN European Organization for Nuclear Research)
Autism Spectrum Disorder Research
preprint

DEEP SYSTEMISING AS EVOLUTIONARILY RETAINED COGNITIVE ARCHITECTURE: Causal-Depth Processing as a Qualitatively Distinct Architecture in High-Systemising Autistic Profiles Across Neurological, Evolutionary, Psychological, and Organisational Levels of

Matthew Richard Hodgson
preprint en

Abstract

This paper proposes a cross-level theoretical account of high-systemising autistic cognition. The central claim is that autistic systemising does not simply occur more frequently than neurotypical systemising — it terminates at a different level. Neurotypical systemising stops at the surface pattern sufficient for social coordination; autistic causal-depth systemising continues to the underlying causal logic that makes the pattern necessary. That distinction — a difference of kind rather than degree — is the framework's central theoretical proposition. The argument develops across four levels of analysis: Neurological: reduced thalamic gating efficiency, arising from excitatory-inhibitory imbalance and aberrant precision weighting, produces a system in which prior beliefs are insufficiently weighted to halt processing at the template level. Evolutionary: specific Neanderthal-derived variants enriched in autistic individuals (Pauly et al., 2024), interpreted as consistent with a retained ancestral processing style maintained through de novo regeneration and episodic frequency-dependent selective value. Psychological: causal-depth processing generates characteristic costs (generative masking, rest-resistant burnout, HPA axis amplification) and outputs (first-principles problem-solving, structural identification of system failure). Sociological: the architecture is proposed to function as a structural catalyst at institutional change points, at characteristic personal cost. A processing-budget construct is developed to explain within-population variation in observable systemising, identifying a clinically significant, budget-depleted subpopulation that is indistinguishable from architectural non-systemisers on current instruments. Nine testable hypotheses (H1–H9) are generated, together with a four-phase empirical research programme gated on the development of a validated processing-depth instrument. The paper is a theoretical synthesis rather than an empirical study. All citations have been verified against primary sources; inferential extensions beyond the cited evidence are flagged explicitly throughout. This is a pre-print made available for scholarly engagement prior to peer review.

Zenodo (CERN European Organization for Nuclear Research)
Autism Spectrum Disorder Research
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DEEP SYSTEMISING AS EVOLUTIONARILY RETAINED COGNITIVE ARCHITECTURE: Causal-Depth Processing as a Qualitatively Distinct Architecture in High-Systemising Autistic Profiles Across Neurological, Evolutionary, Psychological, and Organisational Levels of — Matthew Richard Hodgson · Zenodo (CERN European Organization for Nuclear Research) (2026) | TGRS Research Map | TGRS