<scp>CRYAB</scp> is compromised in pathologically affected oligodendrocytes in multiple system atrophy
CRYAB is hypermethylated and colocalizes with α-synuclein in multiple system atrophy oligodendrocytes. The authors declare no conflicts of interest. The data that support the findings of this study are available from the corresponding authors upon reasonable request.
Authors
- Woojin Scott Kim (ORCID: https://orcid.org/0000-0002-4707-933X)
- Felicia Suteja
- Jen-Hsiang T. Hsiao (ORCID: https://orcid.org/0000-0003-4933-0417)
- Shrey Shahdadpuri
- Finula I. Isik (ORCID: https://orcid.org/0000-0003-2060-1180)
- Nicolas Dzamko (ORCID: https://orcid.org/0000-0002-9121-0294)
- YuHong Fu (ORCID: https://orcid.org/0000-0003-4539-2039)
- Glenda M. Halliday (ORCID: https://orcid.org/0000-0003-0422-8398)
Institutions
- The University of Sydney (AU)
- UNSW Sydney (AU)
Publication Details
- Journal
- Brain Pathology
- Published
- 2026-05-28
- DOI
- https://doi.org/10.1111/bpa.70112
- Primary Topic
- Neurogenesis and neuroplasticity mechanisms
- Type
- article
- Field-Weighted Citation Impact
- 0.00
Funders
- National Ataxia Foundation