Genes, other than Muc5b, are associated with bleomycin-induced lung injury

The MUC5B promoter variant rs35705950 is the dominant risk factor for the development of idiopathic pulmonary fibrosis (IPF), yet its low penetrance remains unexplained. All eight founder strains of the Diversity Outbred (DO) mouse population carry the risk allele at the orthologous site, yet exhibited variable bleomycin-induced MUC5B expression and fibrosis severity. Herein, using the DO bleomycin model, we find that the Muc5b locus regulates MUC5B expression at 3 weeks, and at 10 weeks, several quantitative trait loci (QTLs) are associated with radiologic features of lung fibrosis, such as parenchymal consolidations. Cross-species integrations with human IPF studies reveal heterogeneous epithelial patterns and variable correlation with MUC5B among orthologous QTL genes, and expression of novel proteins in pathognomonic lesions of IPF. This study establishes a radiomics-genetics resource generated from DO mice subjected to bleomycin-induced lung injury, enabling systematic dissection of genetically driven heterogeneity in addition to MUC5B in lung imaging traits.

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Journal
Communications Biology
Published
2026-05-12
DOI
https://doi.org/10.1038/s42003-026-10239-3
Primary Topic
Interstitial Lung Diseases and Idiopathic Pulmonary Fibrosis
Type
article
Field-Weighted Citation Impact
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article

Genes, other than Muc5b, are associated with bleomycin-induced lung injury

Gregory R. Keele, Evgenia Dobrinskikh, Dana A. Godfrey, Kristina Hatakka et al.
Communications Biology
Interstitial Lung Diseases and Idiopathic Pulmonary Fibrosis
article

Genes, other than Muc5b, are associated with bleomycin-induced lung injury

Gregory R. Keele, Evgenia Dobrinskikh, Dana A. Godfrey, Kristina Hatakka, Yu Wang, Corinne Hennessy, David A. Schwartz, Cheryl L. Ackert-Bicknell, Stephen M. Humphries, Anna Peljto, Samir N. P. Kelada, David E. Clouthier, Jonathan Cardwell, Ivana V. Yang
article en

Abstract

The MUC5B promoter variant rs35705950 is the dominant risk factor for the development of idiopathic pulmonary fibrosis (IPF), yet its low penetrance remains unexplained. All eight founder strains of the Diversity Outbred (DO) mouse population carry the risk allele at the orthologous site, yet exhibited variable bleomycin-induced MUC5B expression and fibrosis severity. Herein, using the DO bleomycin model, we find that the Muc5b locus regulates MUC5B expression at 3 weeks, and at 10 weeks, several quantitative trait loci (QTLs) are associated with radiologic features of lung fibrosis, such as parenchymal consolidations. Cross-species integrations with human IPF studies reveal heterogeneous epithelial patterns and variable correlation with MUC5B among orthologous QTL genes, and expression of novel proteins in pathognomonic lesions of IPF. This study establishes a radiomics-genetics resource generated from DO mice subjected to bleomycin-induced lung injury, enabling systematic dissection of genetically driven heterogeneity in addition to MUC5B in lung imaging traits.

Communications Biology
University of North Carolina at Chapel Hill (US), RTI International (US), National Jewish Health (US), Children's Hospital Colorado (US), Rocky Mountain MS Center (US), University of Colorado Anschutz Medical Campus (US), University of Colorado Denver (US)
National Heart, Lung, and Blood Institute
Good health and well-being
Openalex Percentile: Top 5%
Interstitial Lung Diseases and Idiopathic Pulmonary Fibrosis
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