Pan-cancer neurotransmitter receptor alterations define neuroregulatory subtypes with prognostic significance

Cancer neuroscience highlights the critical role of neural signaling in tumors, yet a pan-cancer understanding of neuroregulatory dysregulation is lacking. We systematically characterized 130 neurotransmitter receptor (NTR) genes across 9,125 tumors from 33 cancer types. Our analysis revealed heterogeneous NTR mutations, with 17 cancer types showing elevated rates. Notably, amplification of the muscarinic receptor gene CHRM2 exhibited mutual exclusivity with the clinically actionable gene ERBB2, suggesting its potential as a therapeutic target. NTR expression was widely dysregulated and associated with altered DNA methylation, microRNA (miRNA) expression, and patient prognosis. Unsupervised clustering identified five recurrent neuroregulatory subtypes with distinct clinical and molecular features across cancers. Using low-grade glioma and liver cancer as examples, we validated that the S4 and S1 subtypes were consistently correlated with aggressive disease and poor outcomes in these two cancers, respectively. This study establishes a foundational framework for advancing cancer neuroscience and targeting neuroregulatory signaling in cancer therapy.

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Publication Details

Journal
Cell Reports
Published
2026-05-01
DOI
https://doi.org/10.1016/j.celrep.2026.117340
Primary Topic
Cancer, Stress, Anesthesia, and Immune Response
Type
article
Field-Weighted Citation Impact
0.00

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article

Pan-cancer neurotransmitter receptor alterations define neuroregulatory subtypes with prognostic significance

Shangyi Luo, Xin Hao, Zhuang Xiong, Yajing Zhang et al.
Cell Reports
Cancer, Stress, Anesthesia, and Immune Response
article

Pan-cancer neurotransmitter receptor alterations define neuroregulatory subtypes with prognostic significance

Shangyi Luo, Xin Hao, Zhuang Xiong, Yajing Zhang, Xue Zhang, Lei Guo, Li Liu, Shupei Qiao
article en

Abstract

Cancer neuroscience highlights the critical role of neural signaling in tumors, yet a pan-cancer understanding of neuroregulatory dysregulation is lacking. We systematically characterized 130 neurotransmitter receptor (NTR) genes across 9,125 tumors from 33 cancer types. Our analysis revealed heterogeneous NTR mutations, with 17 cancer types showing elevated rates. Notably, amplification of the muscarinic receptor gene CHRM2 exhibited mutual exclusivity with the clinically actionable gene ERBB2, suggesting its potential as a therapeutic target. NTR expression was widely dysregulated and associated with altered DNA methylation, microRNA (miRNA) expression, and patient prognosis. Unsupervised clustering identified five recurrent neuroregulatory subtypes with distinct clinical and molecular features across cancers. Using low-grade glioma and liver cancer as examples, we validated that the S4 and S1 subtypes were consistently correlated with aggressive disease and poor outcomes in these two cancers, respectively. This study establishes a foundational framework for advancing cancer neuroscience and targeting neuroregulatory signaling in cancer therapy.

Cell ReportsVol. 45(5)
Fujian Medical University (CN), Harbin Medical University (CN), Central China Normal University (CN), Heilongjiang University (CN), Fuzhou University (CN)
National Natural Science Foundation of China, China Postdoctoral Science Foundation, Natural Science Foundation of Fujian Province
Good health and well-being
Openalex Percentile: Top 8%
Cancer, Stress, Anesthesia, and Immune Response
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