Transcriptional and epigenetic plasticity drive an alternative non-clonal mechanism of resistance to KRASG12D inhibition in pancreatic cancer
Transcriptional and epigenetic plasticity drive an alternative non-clonal mechanism of resistance to KRASG12D inhibition in pancreatic cancer
Authors
- Stefano Ugel (ORCID: https://orcid.org/0000-0002-6639-7608)
- Carmine Carbone (ORCID: https://orcid.org/0000-0001-5168-747X)
- Lorenzo Priori
- Alessia Caggiano
- Claudio Sette (ORCID: https://orcid.org/0000-0003-2864-8266)
- Vincenzo Corbo (ORCID: https://orcid.org/0000-0002-6340-8009)
- Gian Luca Rampioni Vinciguerra (ORCID: https://orcid.org/0000-0003-0739-1668)
- Geny Piro (ORCID: https://orcid.org/0000-0003-3708-5397)
- Giampaolo Tortora (ORCID: https://orcid.org/0000-0002-1378-4962)
- Vincenzo Bronte (ORCID: https://orcid.org/0000-0002-3741-5141)
- Davide Pasini (ORCID: https://orcid.org/0000-0002-8270-7143)
- Annachiara Esposito (ORCID: https://orcid.org/0000-0002-5017-1954)
- Antonio Agostini (ORCID: https://orcid.org/0000-0002-6528-7140)
- Federica Cinti
- Diego de la Rosa
- Francesco De Sanctis
- Diego Iacuone
- Anna Ceccarelli
Institutions
- Agostino Gemelli University Polyclinic (IT)
Publication Details
- Journal
- Cancer Communications
- Published
- 2026-04-26
- DOI
- https://doi.org/10.34133/cancomm.0030
- Primary Topic
- Protein Kinase Regulation and GTPase Signaling
- Type
- article
- Field-Weighted Citation Impact
- 0.00