Loss of TMEM65 in mice causes mitochondrial disease mediated by mitochondrial Ca2+

Authors

Audrey Noguchi
Alex Shamoun
Yingfan Zhang (ORCID: https://orcid.org/0000-0001-5932-2440)
Angel Aponte (ORCID: https://orcid.org/0000-0001-5684-2715)
Raúl Covián (ORCID: https://orcid.org/0000-0002-0102-9222)
Brian Glancy (ORCID: https://orcid.org/0000-0002-8571-244X)
Laura Reyes
Sachiko Nakamori
Elizabeth Murphy
Jeeva Munasinghe
Junhui Sun
Danielle Springer
Hailey A. Parry
Chengyu Liu

Institutions

National Institutes of Health (US)
National Institute of Arthritis and Musculoskeletal and Skin Diseases (US)
National Institute of Biomedical Imaging and Bioengineering (US)
National Heart, Lung, and Blood Institute (US)
National Institute of Neurological Disorders and Stroke (US)

Publication Details

Journal: Nature Communications
Published: 2026-04-14
DOI: https://doi.org/10.1038/s41467-026-71761-w
Primary Topic: Mitochondrial Function and Pathology
Type: article
Field-Weighted Citation Impact: 0.00

Funders

National Heart, Lung, and Blood Institute

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article

Loss of TMEM65 in mice causes mitochondrial disease mediated by mitochondrial Ca2+

Audrey Noguchi, Alex Shamoun, Yingfan Zhang, Angel Aponte et al.

Apr 14, 2026

Nature Communications

Mitochondrial Function and Pathology

article

Loss of TMEM65 in mice causes mitochondrial disease mediated by mitochondrial Ca2+

Audrey Noguchi, Alex Shamoun, Yingfan Zhang, Angel Aponte, Raúl Covián, Brian Glancy, Laura Reyes, Sachiko Nakamori, Elizabeth Murphy, Jeeva Munasinghe, Junhui Sun, Danielle Springer, Hailey A. Parry, Chengyu Liu

April 14, 2026article en

Abstract

No abstract available for this paper.

Nature Communications

National Institutes of Health (US), National Institute of Arthritis and Musculoskeletal and Skin Diseases (US), National Institute of Biomedical Imaging and Bioengineering (US), National Heart, Lung, and Blood Institute (US), National Institute of Neurological Disorders and Stroke (US)

National Heart, Lung, and Blood Institute

Openalex Percentile: Top 23%

Mitochondrial Function and Pathology

DOI Open Access PDF OpenAlex

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