NO- and S-nitrosothiols-dependent relaxation in large and small resistance blood vessels
influx, KCa channel activation, and EDH-dependent vasodilation independently of cGMP production. Finally, we examine how extravascular components-including perivascular adipose tissue and red blood cells-modulate EDH signaling under physiological and pathological conditions.
Authors
- Annie Beuve (ORCID: https://orcid.org/0000-0003-4389-5388)
- Mauricio A. Lillo (ORCID: https://orcid.org/0000-0002-2823-6619)
Institutions
- Rutgers, The State University of New Jersey (US)
Publication Details
- Journal
- Frontiers in Physiology
- Published
- 2026-04-13
- DOI
- https://doi.org/10.3389/fphys.2026.1801670
- Primary Topic
- Nitric Oxide and Endothelin Effects
- Type
- article
- Field-Weighted Citation Impact
- 0.00
Funders
- American Heart Association
- National Institutes of Health