MicroRNA‐210‐mediated mitochondrial reactive oxygen species contribute to Ca <sub>V</sub> 1.2 channel hyperactivity in uterine arteries of pregnant sheep acclimatized to high‐altitude hypoxia

1.2 hyperactivity in uterine arteries from pregnant ewes. MicroRNA-210 significantly increased phenylephrine-induced vasoconstriction of uterine arteries of pregnant ewes, an effect that was inhibited by MitoQ.

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Publication Details

Journal
The Journal of Physiology
Published
2026-04-11
DOI
https://doi.org/10.1113/jp290989
Primary Topic
Pregnancy and preeclampsia studies
Type
article
Field-Weighted Citation Impact
0.00

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article

MicroRNA‐210‐mediated mitochondrial reactive oxygen species contribute to Ca V 1.2 channel hyperactivity in uterine arteries of pregnant sheep acclimatized to high‐altitude hypoxia

Chiranjib Dasgupta, Styliani Goulopoulou, Rui Song, Arlin B. Blood et al.
The Journal of Physiology
Pregnancy and preeclampsia studies
article

MicroRNA‐210‐mediated mitochondrial reactive oxygen species contribute to Ca V 1.2 channel hyperactivity in uterine arteries of pregnant sheep acclimatized to high‐altitude hypoxia

Chiranjib Dasgupta, Styliani Goulopoulou, Rui Song, Arlin B. Blood, Xiang‐Qun Hu, T.M. Liu, Lubo Zhang, Meijuan Zhang, Eugenia MataGreenwood
article en

Abstract

1.2 hyperactivity in uterine arteries from pregnant ewes. MicroRNA-210 significantly increased phenylephrine-induced vasoconstriction of uterine arteries of pregnant ewes, an effect that was inhibited by MitoQ.

The Journal of Physiology
Loma Linda University (US)
National Institutes of Health, National Heart, Lung, and Blood Institute, Eunice Kennedy Shriver National Institute of Child Health and Human Development
Openalex Percentile: Top 24%
Pregnancy and preeclampsia studies
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