Translation in proximity to forming autophagosomes during sustained autophagy

Autophagy is an evolutionarily conserved catabolic process. In a process requiring a cascade of over 35 autophagy-related genes (Atg), a cupped phagophore membrane expands to surround cytoplasmic material, and seals itself to form an autophagosome, which finally fuses with lysosomes. Large numbers of autophagosomes form during stress responses, while simultaneously cells drastically reduce translation to conserve energy. Here, using proximity-labeling and Fluorescence in situ Hybridization we demonstrate that multiple mRNAs encoding proteins required for autophagy preferentially localize in proximity to forming autophagosomes. Polysome fractionation and proteomics of nascent proteins in proximity to forming autophagosomes provides evidence for the local translation of these mRNAs. Translation and the ribosome-binding protein RACK1 were required for the localization of these mRNAs to forming autophagosomes. Inhibition of translation or knockdown of RACK1 caused depletion of several proteins required for autophagy and a reduction in the number of autophagosomes. Local translation may enable a rapid, energy-efficient supply of proteins for autophagy to enable cells to massively induce autophagy while conserving energy during cell stress.

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Publication Details

Journal
Nature Communications
Published
2026-04-10
DOI
https://doi.org/10.1038/s41467-026-71551-4
Primary Topic
Autophagy in Disease and Therapy
Type
article
Field-Weighted Citation Impact
0.00

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article

Translation in proximity to forming autophagosomes during sustained autophagy

Ryan C. Russell, Kaela O’Connor, Alexandre Savard, Yunping Xue et al.
Nature Communications
Autophagy in Disease and Therapy
article

Translation in proximity to forming autophagosomes during sustained autophagy

Ryan C. Russell, Kaela O’Connor, Alexandre Savard, Yunping Xue, Karsten Nalbach, Christian Behrends, Derrick Gibbings, Karyn E. King
article en

Abstract

Autophagy is an evolutionarily conserved catabolic process. In a process requiring a cascade of over 35 autophagy-related genes (Atg), a cupped phagophore membrane expands to surround cytoplasmic material, and seals itself to form an autophagosome, which finally fuses with lysosomes. Large numbers of autophagosomes form during stress responses, while simultaneously cells drastically reduce translation to conserve energy. Here, using proximity-labeling and Fluorescence in situ Hybridization we demonstrate that multiple mRNAs encoding proteins required for autophagy preferentially localize in proximity to forming autophagosomes. Polysome fractionation and proteomics of nascent proteins in proximity to forming autophagosomes provides evidence for the local translation of these mRNAs. Translation and the ribosome-binding protein RACK1 were required for the localization of these mRNAs to forming autophagosomes. Inhibition of translation or knockdown of RACK1 caused depletion of several proteins required for autophagy and a reduction in the number of autophagosomes. Local translation may enable a rapid, energy-efficient supply of proteins for autophagy to enable cells to massively induce autophagy while conserving energy during cell stress.

Nature Communications
University of Ottawa (CA), Munich Cluster for Systems Neurology (DE), Ludwig-Maximilians-Universität München (DE)
Canadian Institutes of Health Research
Affordable and clean energy
Openalex Percentile: Top 15%
Autophagy in Disease and Therapy
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