Editorial Note: Endoplasmic reticulum stress-induced autophagy provides cytoprotection from chemical hypoxia and oxidant injury and ameliorates renal ischemia-reperfusion injury

Endoplasmic reticulum stress-induced autophagy provides cytoprotection from chemical hypoxia and oxidant injury and ameliorates renal

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Publication Details

Journal
PLoS ONE
Published
2026-04-02
DOI
https://doi.org/10.1371/journal.pone.0346190
Primary Topic
Autophagy in Disease and Therapy
Type
article
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0.00
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article

Editorial Note: Endoplasmic reticulum stress-induced autophagy provides cytoprotection from chemical hypoxia and oxidant injury and ameliorates renal ischemia-reperfusion injury

The PLOS One Editors
PLoS ONE
Autophagy in Disease and Therapy
article

Editorial Note: Endoplasmic reticulum stress-induced autophagy provides cytoprotection from chemical hypoxia and oxidant injury and ameliorates renal ischemia-reperfusion injury

The PLOS One Editors
article en

Abstract

Endoplasmic reticulum stress-induced autophagy provides cytoprotection from chemical hypoxia and oxidant injury and ameliorates renal

PLoS ONEVol. 21(4)
Zero hunger
Openalex Percentile: Top 32%
Autophagy in Disease and Therapy
DOIOpen Access PDFOpenAlex
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Editorial Note: Endoplasmic reticulum stress-induced autophagy provides cytoprotection from chemical hypoxia and oxidant injury and ameliorates renal ischemia-reperfusion injury — The PLOS One Editors · PLoS ONE (2026) | TGRS Research Map | TGRS