<scp>BRAP</scp> Homologue Deficiency Exacerbates Airway Inflammation in Mouse Models of Experimental Asthma‐Like Disease
mice may promote increased TSLP release from airway epithelial cells, thereby contributing to the exacerbation of allergic inflammation and asthma pathogenesis. Collectively, these results indicate a role for BRAP in maintaining epithelial barrier function during allergic asthma.
Authors
- H. Christian Weber (ORCID: https://orcid.org/0000-0001-9005-6988)
- Xiangping Qu (ORCID: https://orcid.org/0000-0003-2794-168X)
- Rujiao Liu (ORCID: https://orcid.org/0000-0002-6803-0594)
- Jiaoyun Zheng
- Hao Wang (ORCID: https://orcid.org/0000-0001-6881-9977)
- Wenrui Zhang (ORCID: https://orcid.org/0000-0002-0223-1924)
- Yi-Xiang Wang (ORCID: https://orcid.org/0000-0002-5246-5497)
Institutions
- Boston University (US)
- Central South University (CN)
- Zhuhai People's Hospital (CN)
- Hunan Vocational Institute of Technology (CN)
- Hunan Vocational College of Safety Technology (CN)
- Second Xiangya Hospital of Central South University (CN)
Publication Details
- Journal
- Clinical and Experimental Pharmacology and Physiology
- Published
- 2026-04-01
- DOI
- https://doi.org/10.1111/1440-1681.70119
- Primary Topic
- Asthma and respiratory diseases
- Type
- article
- Field-Weighted Citation Impact
- 0.00
Funders
- National Natural Science Foundation of China
- Natural Science Foundation of Hunan Province