Cancer behaves like a virus – 11 molecular identities + 12 dual-action substances

Oncogenic viruses (EBV, HCV, HBV, HPV, HHV-8, HTLV-1, H. pylori) produce the same mechanisms as common viruses: inflammation, coagulation, biofilm, NF-κB activation, hypoxia, iron addiction, Warburg effect, local immunosuppression. The difference is not in the mechanism but in the time course: acute/visible vs chronic/silent. Twelve substances (aspirin, curcumin, EGCG, artemisinin, berberine, quercetin, sulforaphane, ivermectin, fenbendazole, ursolic acid, resveratrol, lactoferrin) target the same molecular pathways in both viruses and tumors. Aspirin blocks inflammation, coagulation, and biofilm, reducing the incidence of 7 cancers – with a 52% reduction in hepatocellular carcinoma in patients with chronic viral hepatitis (HR=0.48, p<0.001). If cancer were purely genetic, these substances would not work on both. They do, because the tumor is the chronic consequence of the virus.