<scp>IL</scp> ‐33 Drives Inflammatory Changes and Extracellular Trap Formation in Eosinophils Involving Oxidised <scp>LDL</scp> and Complement Pathways

IL-33, in conjunction with oxLDL and the complement cascade, induces inflammatory changes in eosinophils, promoting an ETosis-prone phenotype. These pathways represent potential therapeutic targets in refractory eosinophilic diseases.

Institutions

Publication Details

Journal
Allergy
Published
2026-03-24
DOI
https://doi.org/10.1111/all.70313
Primary Topic
IL-33, ST2, and ILC Pathways
Type
article
Field-Weighted Citation Impact
0.00
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article

IL ‐33 Drives Inflammatory Changes and Extracellular Trap Formation in Eosinophils Involving Oxidised LDL and Complement Pathways

Allergy
IL-33, ST2, and ILC Pathways
article

IL ‐33 Drives Inflammatory Changes and Extracellular Trap Formation in Eosinophils Involving Oxidised LDL and Complement Pathways

article en

Abstract

IL-33, in conjunction with oxLDL and the complement cascade, induces inflammatory changes in eosinophils, promoting an ETosis-prone phenotype. These pathways represent potential therapeutic targets in refractory eosinophilic diseases.

Allergy
Akita University (JP), Keio University (JP), Yokohama City University Medical Center (JP), Kazusa DNA Research Institute (JP), RIKEN Center for Integrative Medical Sciences (JP), National Defense Medical College (JP), Yokohama City University (JP)
Openalex Percentile: Top 10%
IL-33, ST2, and ILC Pathways
DOIOpen Access PDFOpenAlex
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<scp>IL</scp> ‐33 Drives Inflammatory Changes and Extracellular Trap Formation in Eosinophils Involving Oxidised <scp>LDL</scp> and Complement Pathways · Allergy (2026) | TGRS Research Map | TGRS