The Potential Molecular Mechanisms and Key Genes of Early Deterioration of Parenchymal Lesions in Cerebral Venous Sinus Thrombosis

It was shown that focal adhesion and ECM-receptor interaction may be involved in the mechanisms of EDPL, and RELN, CAV1, CCNB1, and NME1-NME2 may be promising intervention targets for EDPL.

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Publication Details

Journal
Current Neurovascular Research
Published
2026-03-16
DOI
https://doi.org/10.2174/0115672026438053260120052902
Primary Topic
Cerebral Venous Sinus Thrombosis
Type
article
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article

The Potential Molecular Mechanisms and Key Genes of Early Deterioration of Parenchymal Lesions in Cerebral Venous Sinus Thrombosis

Zhijian Liang, Jin Li, Jun Wang, Ya Chen et al.
Current Neurovascular Research
Cerebral Venous Sinus Thrombosis
article

The Potential Molecular Mechanisms and Key Genes of Early Deterioration of Parenchymal Lesions in Cerebral Venous Sinus Thrombosis

Zhijian Liang, Jin Li, Jun Wang, Ya Chen, Gengyu Cen
article en

Abstract

It was shown that focal adhesion and ECM-receptor interaction may be involved in the mechanisms of EDPL, and RELN, CAV1, CCNB1, and NME1-NME2 may be promising intervention targets for EDPL.

Current Neurovascular ResearchVol. 23
First Affiliated Hospital of GuangXi Medical University (CN)
Openalex Percentile: Top 23%
Cerebral Venous Sinus Thrombosis
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