Downregulation of the Long Non-Coding RNA MDL1AS Alters Metabolism, Differentiation, and Radiosensitivity in NTERA2 and SH-SY5Y Cells

<b>Background/Objectives</b>: Non-coding RNAs provide new chances of targeting multiple oncogenic pathways. Many long non-coding RNAs (lncRNAs) are being characterized as relevant in cancer initiation, progression, and recurrence. <i>Mitochondrial D-loop 1 antisense lncRNA (MDL1AS)</i> is a novel lncRNA that might be important in cancer development, so the aim of this project was to understand its function in differently differentiated cancer cells. <b>Methods</b>: The effects of <i>MDL1AS</i> downregulation on the cellular behavior of NTERA2 and SH-SY5Y cell lines were studied. <b>Results</b>: <i>MDL1AS</i> reduction inhibited oxidative phosphorylation in NTERA2 cells and induced neuritic differentiation in SH-SY5Y cells. This downregulation also produced a strong DNA damage response (DDR) and an increased apoptotic signature by RNAseq analysis, and decreased proliferation in both cell lines. It also decreased radiosensitivity in NTERA2 cells but not in SH-SY5Y. <b>Conclusions</b>: These results suggest that <i>MDL1AS</i> reduction can modulate radiosensitivity, metabolism, and differentiation in a cell type-specific manner. Furthermore, <i>MDL1AS</i> may constitute a predictive biomarker and a molecular target for new therapies.

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Journal
Cancers
Published
2026-03-12
DOI
https://doi.org/10.3390/cancers18060928
Primary Topic
Cancer-related molecular mechanisms research
Type
article
Field-Weighted Citation Impact
0.00

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article

Downregulation of the Long Non-Coding RNA MDL1AS Alters Metabolism, Differentiation, and Radiosensitivity in NTERA2 and SH-SY5Y Cells

Pablo Garrido, Adrián Casas-Benito, J. Alfredo Martínéz
Cancers
Cancer-related molecular mechanisms research
article

Downregulation of the Long Non-Coding RNA MDL1AS Alters Metabolism, Differentiation, and Radiosensitivity in NTERA2 and SH-SY5Y Cells

Pablo Garrido, Adrián Casas-Benito, J. Alfredo Martínéz
article en

Abstract

<b>Background/Objectives</b>: Non-coding RNAs provide new chances of targeting multiple oncogenic pathways. Many long non-coding RNAs (lncRNAs) are being characterized as relevant in cancer initiation, progression, and recurrence. <i>Mitochondrial D-loop 1 antisense lncRNA (MDL1AS)</i> is a novel lncRNA that might be important in cancer development, so the aim of this project was to understand its function in differently differentiated cancer cells. <b>Methods</b>: The effects of <i>MDL1AS</i> downregulation on the cellular behavior of NTERA2 and SH-SY5Y cell lines were studied. <b>Results</b>: <i>MDL1AS</i> reduction inhibited oxidative phosphorylation in NTERA2 cells and induced neuritic differentiation in SH-SY5Y cells. This downregulation also produced a strong DNA damage response (DDR) and an increased apoptotic signature by RNAseq analysis, and decreased proliferation in both cell lines. It also decreased radiosensitivity in NTERA2 cells but not in SH-SY5Y. <b>Conclusions</b>: These results suggest that <i>MDL1AS</i> reduction can modulate radiosensitivity, metabolism, and differentiation in a cell type-specific manner. Furthermore, <i>MDL1AS</i> may constitute a predictive biomarker and a molecular target for new therapies.

CancersVol. 18(6)
Center for Biomedical Research of La Rioja (ES)
European Regional Development Fund
Good health and well-being
Openalex Percentile: Top 24%
Cancer-related molecular mechanisms research
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