DLD, upregulated by YY1, fuels glioblastoma multiforme progression via EphA2 phosphorylation to activate PI3K/AKT/mTOR
DLD functions as an oncogene in GBM and represents a potential biomarker and therapeutic target.
Authors
- Yan Zhou (ORCID: https://orcid.org/0000-0001-6992-6491)
- Min Yuan (ORCID: https://orcid.org/0000-0002-1382-9171)
- Jun Su (ORCID: https://orcid.org/0000-0002-9349-8199)
- Jia You (ORCID: https://orcid.org/0000-0001-8864-1512)
- Guodong Tang (ORCID: https://orcid.org/0000-0003-1994-8785)
- Zijin Zhao
- Haoyu Li
- Wei Huang
Institutions
- Central South University (CN)
- The First People's Hospital of Changde (CN)
- National Clinical Research (US)
- Hunan Children's Hospital (CN)
- Hunan Entry-Exit Inspection and Quarantine Bureau (CN)
- Zhuzhou Central Hospital (CN)
- Xiangya Hospital Central South University (CN)
Publication Details
- Journal
- QJM
- Published
- 2026-03-04
- DOI
- https://doi.org/10.1093/qjmed/hcag070
- Primary Topic
- Axon Guidance and Neuronal Signaling
- Type
- article
- Field-Weighted Citation Impact
- 0.00