Salvianolic acid B attenuates post-cardiac arrest cerebral ischemia–reperfusion injury via activation of the Nrf2 signaling pathway
SalB protects against CI/R-induced neurological injury by reducing oxidative stress, inflammation, and apoptosis through Nrf2 activation, supporting its potential as a therapeutic agent for post-CA cerebral injury.
Authors
- Shasha Yu (ORCID: https://orcid.org/0000-0002-6303-1387)
- Bo Feng (ORCID: https://orcid.org/0000-0001-5084-6715)
- Guangsheng Guo
- Chuanbao Lv
Institutions
- Harbin Medical University (CN)
- Beijing United Family Hospital (CN)
- First Affiliated Hospital of Guangzhou University of Chinese Medicine (CN)
- First Affiliated Hospital of Harbin Medical University (CN)
- 421 Hospital of PLA (CN)
Publication Details
- Journal
- European journal of medical research
- Published
- 2026-03-09
- DOI
- https://doi.org/10.1186/s40001-026-04177-3
- Primary Topic
- Neuroinflammation and Neurodegeneration Mechanisms
- Type
- article
- Field-Weighted Citation Impact
- 0.00