<scp>LIN28A</scp> blocks the <scp>EndMT</scp> process of high glucose‐induced <scp>HRMECs</scp> by stabilizing <scp>SIRT6 mRNA</scp>

The stabilization of SIRT6 mRNA by LIN28A activated AMPK signaling, inhibiting the EndMT process in HRMECs caused by HG.

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Publication Details

Journal
Journal of Diabetes Investigation
Published
2026-03-07
DOI
https://doi.org/10.1111/jdi.70284
Primary Topic
Endoplasmic Reticulum Stress and Disease
Type
article
Field-Weighted Citation Impact
0.00
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article

LIN28A blocks the EndMT process of high glucose‐induced HRMECs by stabilizing SIRT6 mRNA

Yali Nan, Z D Zhang, Zixu Huang, Zongming Song et al.
Journal of Diabetes Investigation
Endoplasmic Reticulum Stress and Disease
article

LIN28A blocks the EndMT process of high glucose‐induced HRMECs by stabilizing SIRT6 mRNA

Yali Nan, Z D Zhang, Zixu Huang, Zongming Song, Xiaohui Kuang, Xueying Wang
article en

Abstract

The stabilization of SIRT6 mRNA by LIN28A activated AMPK signaling, inhibiting the EndMT process in HRMECs caused by HG.

Journal of Diabetes Investigation
Zhengzhou Central Hospital (CN), Henan Provincial Eye Hospital (CN)
Openalex Percentile: Top 31%
Endoplasmic Reticulum Stress and Disease
DOIOpen Access PDFOpenAlex
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<scp>LIN28A</scp> blocks the <scp>EndMT</scp> process of high glucose‐induced <scp>HRMECs</scp> by stabilizing <scp>SIRT6 mRNA</scp> — Yali Nan, Z D Zhang, et al. · Journal of Diabetes Investigation (2026) | TGRS Research Map | TGRS