Clonal haematopoiesis and risk of incident rheumatoid arthritis: results from the UK biobank

Common CHIP mutations, including TET2, TP53, and SF3B1 mutations, were not associated with risk of incident RA when restricted to those with most complete general outpatient and hospital record follow-up in the UK Biobank.

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Publication Details

Journal
Clinical and Experimental Rheumatology
Published
2025-09-21
DOI
https://doi.org/10.55563/clinexprheumatol/23hcbl
Primary Topic
Rheumatoid Arthritis Research and Therapies
Type
article
Field-Weighted Citation Impact
0.00

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article

Clonal haematopoiesis and risk of incident rheumatoid arthritis: results from the UK biobank

Emily G. Oakes, Jing Cui, M M Uddin, Abhishek Niroula et al.
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Clonal haematopoiesis and risk of incident rheumatoid arthritis: results from the UK biobank

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article en

Abstract

Common CHIP mutations, including TET2, TP53, and SF3B1 mutations, were not associated with risk of incident RA when restricted to those with most complete general outpatient and hospital record follow-up in the UK Biobank.

Clinical and Experimental Rheumatology
Broad Institute (US), Brigham and Women's Hospital (US), Harvard University (US), Massachusetts Institute of Technology (US), University of Gothenburg (SE)
National Institutes of Health
Openalex Percentile: Top 22%
Rheumatoid Arthritis Research and Therapies
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