Rare genetic variant risks in patients with sepsis-associated acute respiratory distress syndrome

Acute respiratory distress syndrome (ARDS) is a complex, heterogeneous, and deadly condition often resulting from pulmonary lesions due to sepsis, among other causes. There is a lack of targeted therapies to specifically treat the patients. Common genetic factors in the population (frequency > 1%) have been associated with ARDS susceptibility, but systematic genetic screens of the role of rare genetic variants are lacking. We used the network of known molecular interactions to identify ARDS risks from clusters of biologically related genes containing qualifying variants (QVs) with frequency < 1% likely affecting function. We conducted whole-exome sequencing in sepsis patients from the GEN-SEP cohort (n = 822, of which 272 developed ARDS). A network-based heterogeneity clustering algorithm was used to discover significant gene clusters (p < 1 × 10–5). Gene-set enrichment analysis and logistic regression models aggregating QVs were used for cross-verification to confirm consistency and deepen understanding of the effect sizes of gene clusters. We identified 19 significant clusters (plowest = 3.29 × 10–10), each containing an average of 102 genes (11.6% mean similarity). QVs in nine gene clusters were associated with sepsis-associated ARDS (plowest = 1 × 10–5) but were not associated with 28-day survival. Clusters were enriched in several biological pathways, notably the Toll-like receptor cascades. These results support a marked genetic heterogeneity underlying ARDS susceptibility and the presence of rare risk variants involving multiple biological processes that are associated with sepsis outcomes. Particularly, they underscore the importance of rare variants in genes of the Toll-like receptor cascades in the risk for sepsis-associated ARDS.

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Publication Details

Journal
Respiratory Research
Published
2026-02-25
DOI
https://doi.org/10.1186/s12931-026-03588-4
Primary Topic
Sepsis Diagnosis and Treatment
Type
article
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article

Rare genetic variant risks in patients with sepsis-associated acute respiratory distress syndrome

Jesus Blanco, Eva Suarez-Pajes, Elena Espinosa, Tamara Hernández-Beeftink et al.
Respiratory Research
Sepsis Diagnosis and Treatment
article

Rare genetic variant risks in patients with sepsis-associated acute respiratory distress syndrome

Jesus Blanco, Eva Suarez-Pajes, Elena Espinosa, Tamara Hernández-Beeftink, Eva Tosco-Herrera, Luis A. Rubio‐Rodríguez, Aitana Alonso-González, M. Almudena Sánchez Martín, Demetrio Carriedo, Miryam Prieto-González, Jesús Monterrubio Villar, J M Añón, David Domínguez, Adrián Muñoz-Barrera, David Jáspez, Carlos Flores, Elena González-Higueras, Itahisa Marcelino-Rodríguez, Beatriz Guillén-Guio, Almudena Corrales, Carlos Rodríguez-Gallego, José M. Lorenzo-Salazar
article en

Abstract

Acute respiratory distress syndrome (ARDS) is a complex, heterogeneous, and deadly condition often resulting from pulmonary lesions due to sepsis, among other causes. There is a lack of targeted therapies to specifically treat the patients. Common genetic factors in the population (frequency > 1%) have been associated with ARDS susceptibility, but systematic genetic screens of the role of rare genetic variants are lacking. We used the network of known molecular interactions to identify ARDS risks from clusters of biologically related genes containing qualifying variants (QVs) with frequency < 1% likely affecting function. We conducted whole-exome sequencing in sepsis patients from the GEN-SEP cohort (n = 822, of which 272 developed ARDS). A network-based heterogeneity clustering algorithm was used to discover significant gene clusters (p < 1 × 10–5). Gene-set enrichment analysis and logistic regression models aggregating QVs were used for cross-verification to confirm consistency and deepen understanding of the effect sizes of gene clusters. We identified 19 significant clusters (plowest = 3.29 × 10–10), each containing an average of 102 genes (11.6% mean similarity). QVs in nine gene clusters were associated with sepsis-associated ARDS (plowest = 1 × 10–5) but were not associated with 28-day survival. Clusters were enriched in several biological pathways, notably the Toll-like receptor cascades. These results support a marked genetic heterogeneity underlying ARDS susceptibility and the presence of rare risk variants involving multiple biological processes that are associated with sepsis outcomes. Particularly, they underscore the importance of rare variants in genes of the Toll-like receptor cascades in the risk for sepsis-associated ARDS.

Respiratory Research
Universidad de Las Palmas de Gran Canaria (ES), St. Michael's Hospital (CA), University of Leicester (GB), Universidad de La Laguna (ES), Hospital Universitario de Canarias (ES), Hospital Universitario La Paz (ES), Instituto de Salud Carlos III (ES), University Hospitals of Leicester NHS Trust (GB), Hospital Universitario Nuestra Señora de Candelaria (ES), Universidad Fernando Pessoa Canarias (ES), Hospital Virgen de la Luz (ES), Hospital Universitario de Gran Canaria Doctor Negrín (ES), Centro de Investigación Biomédica en Red de Enfermedades Respiratorias (ES), Instituto Tecnológico y de Energías Renovables (ES), Hospital General Universitario de Ciudad Real (ES), Hospital Universitario Río Hortega (ES), Hospital Universitario de León (ES), Complejo Asistencial Universitario de Palencia (ES)
Good health and well-being
Openalex Percentile: Top 44%
Sepsis Diagnosis and Treatment
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