Sensing and Reprogramming Surface Receptor Activation With Synthetic Transcriptional Circuits

Cells rely on surface receptors to sense and initiate signalling cascades essential for numerous cellular processes, but engineering of synthetic genetic circuits to sense and rewire receptor activities for user-defined cellular functions remains a challenge. Here we report a synthetic receptor-signalling induced transcription (RESIT) circuit that enables sensing and reprogramming membrane-localized receptor activation to pre-defined transcriptional programs. The RESIT system is designed based on receptor activation mediated split protease complementation and release of membrane-tethered synthetic transcriptional modules. We show that RESIT design is generally applicable to different transcriptional factors and various split viral proteases. This system is further engineered to probe Ca<sup>2+</sup> entry accompanied with PIEZO1 induction and T cell activation, to detect oncogenic receptor tyrosine kinase (RTK) activities and to assess Ras activation proximal to plasma membranes. The versatility of RESIT system is repurposed to actuate diverse therapeutic functions including apoptosis induction, target protein degradation and T cell activation in cells with high RTK activities. The modularity and versatility of RESIT highlight its promise for interrogating juxtamembrane biochemical signaling and rewiring receptor activation to therapeutic functions.

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Publication Details

Journal
Advanced Science
Published
2026-02-23
DOI
https://doi.org/10.1002/advs.202522557
Primary Topic
Phagocytosis and Immune Regulation
Type
article
Field-Weighted Citation Impact
0.00
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article

Sensing and Reprogramming Surface Receptor Activation With Synthetic Transcriptional Circuits

Jun Jiang, Fenglin Wang, F. Liu, Xia Chu et al.
Advanced Science
Phagocytosis and Immune Regulation
article

Sensing and Reprogramming Surface Receptor Activation With Synthetic Transcriptional Circuits

Jun Jiang, Fenglin Wang, F. Liu, Xia Chu, Mei Yuan, Li‐Juan Tang
article en

Abstract

Cells rely on surface receptors to sense and initiate signalling cascades essential for numerous cellular processes, but engineering of synthetic genetic circuits to sense and rewire receptor activities for user-defined cellular functions remains a challenge. Here we report a synthetic receptor-signalling induced transcription (RESIT) circuit that enables sensing and reprogramming membrane-localized receptor activation to pre-defined transcriptional programs. The RESIT system is designed based on receptor activation mediated split protease complementation and release of membrane-tethered synthetic transcriptional modules. We show that RESIT design is generally applicable to different transcriptional factors and various split viral proteases. This system is further engineered to probe Ca<sup>2+</sup> entry accompanied with PIEZO1 induction and T cell activation, to detect oncogenic receptor tyrosine kinase (RTK) activities and to assess Ras activation proximal to plasma membranes. The versatility of RESIT system is repurposed to actuate diverse therapeutic functions including apoptosis induction, target protein degradation and T cell activation in cells with high RTK activities. The modularity and versatility of RESIT highlight its promise for interrogating juxtamembrane biochemical signaling and rewiring receptor activation to therapeutic functions.

Advanced Science
Hunan University (CN)
Openalex Percentile: Top 45%
Phagocytosis and Immune Regulation
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