Astrocyte-microglia crosstalk through Hevin and Toll-like receptor signaling controls developmental thalamocortical synapse refinement

Synapse formation and elimination are two crucial processes that occur concurrently in the developing brain. Astrocytes and microglia control both processes, yet how these two major glial cell types of the central nervous system (CNS) communicate to balance synapse formation and elimination is unknown. Astrocytes secrete the synaptogenic protein Hevin/SPARCL1, which induces the formation and plasticity of thalamocortical synapses in the mouse visual cortex. Here, we found that, in addition to this synaptogenic function, Hevin directly signals to microglia by interacting with Toll-like receptor 4 (TLR4). This signaling occurs when Hevin is proteolytically cleaved, producing a C-terminal fragment that is no longer synaptogenic. We found that Hevin, through TLR4, induces a distinct microglial state defined by increased TLR2 expression and phago-lysosomal content in vitro and in vivo. Microglial TLR4 signaling is required for the proper elimination of thalamocortical synapses during early postnatal development.

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Publication Details

Journal
Neuron
Published
2026-02-01
DOI
https://doi.org/10.1016/j.neuron.2025.12.028
Primary Topic
Neuroinflammation and Neurodegeneration Mechanisms
Type
article
Field-Weighted Citation Impact
0.00
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article

Astrocyte-microglia crosstalk through Hevin and Toll-like receptor signaling controls developmental thalamocortical synapse refinement

Staci D. Bilbo, Crystal Colón Ortiz, Richa Hanamsagar, Justin T Savage et al.
Neuron
Neuroinflammation and Neurodegeneration Mechanisms
article

Astrocyte-microglia crosstalk through Hevin and Toll-like receptor signaling controls developmental thalamocortical synapse refinement

Staci D. Bilbo, Crystal Colón Ortiz, Richa Hanamsagar, Justin T Savage, Jinhu Kim, Carina L. Block, Evelyn J. Hardin, Cagla Eroglu, Juan David Ramírez, Sandeep Kumar Singh, Kristina Sakers
article en

Abstract

Synapse formation and elimination are two crucial processes that occur concurrently in the developing brain. Astrocytes and microglia control both processes, yet how these two major glial cell types of the central nervous system (CNS) communicate to balance synapse formation and elimination is unknown. Astrocytes secrete the synaptogenic protein Hevin/SPARCL1, which induces the formation and plasticity of thalamocortical synapses in the mouse visual cortex. Here, we found that, in addition to this synaptogenic function, Hevin directly signals to microglia by interacting with Toll-like receptor 4 (TLR4). This signaling occurs when Hevin is proteolytically cleaved, producing a C-terminal fragment that is no longer synaptogenic. We found that Hevin, through TLR4, induces a distinct microglial state defined by increased TLR2 expression and phago-lysosomal content in vitro and in vivo. Microglial TLR4 signaling is required for the proper elimination of thalamocortical synapses during early postnatal development.

Neuron
Howard Hughes Medical Institute (US), Duke University (US), Virginia Commonwealth University (US), Duke Medical Center (US), Duke University Hospital (US), Virginia Commonwealth University Medical Center (US)
Openalex Percentile: Top 59%
Neuroinflammation and Neurodegeneration Mechanisms
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Astrocyte-microglia crosstalk through Hevin and Toll-like receptor signaling controls developmental thalamocortical synapse refinement — Staci D. Bilbo, Crystal Colón Ortiz, et al. · Neuron (2026) | TGRS Research Map | TGRS