Mettl3-modified lncRNA Snhg16 aggravates osteoarthritis via promoting chondrocyte ferroptosis by regulating Snd1/Gpx4 axis

Mettl3-mediated m6A modification of Snhg16 facilitated ferroptosis and ECM degradation to aggravate OA process via regulating Snd1/Gpx4 axis, providing a novel target for OA treatment.

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Publication Details

Journal
Connective Tissue Research
Published
2026-02-15
DOI
https://doi.org/10.1080/03008207.2026.2627517
Primary Topic
Ferroptosis and cancer prognosis
Type
article
Field-Weighted Citation Impact
0.00
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article

Mettl3-modified lncRNA Snhg16 aggravates osteoarthritis via promoting chondrocyte ferroptosis by regulating Snd1/Gpx4 axis

X. Ye, Shengtao Chen, Shaobo He, Xiao Fang Yuan et al.
Connective Tissue Research
Ferroptosis and cancer prognosis
article

Mettl3-modified lncRNA Snhg16 aggravates osteoarthritis via promoting chondrocyte ferroptosis by regulating Snd1/Gpx4 axis

X. Ye, Shengtao Chen, Shaobo He, Xiao Fang Yuan, Qiang Li, Liming Shen
article en

Abstract

Mettl3-mediated m6A modification of Snhg16 facilitated ferroptosis and ECM degradation to aggravate OA process via regulating Snd1/Gpx4 axis, providing a novel target for OA treatment.

Connective Tissue ResearchVol. 67(2)
Hunan University (CN)
Clean water and sanitation
Openalex Percentile: Top 54%
Ferroptosis and cancer prognosis
DOISourceOpenAlex
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