Myophosphorylase Knock Out Prevents the Exaggerated Exercise Pressor Reflex in Rats With Simulated Peripheral Artery Disease

Lactate and hydrogen-ions accumulation in contracting myocytes plays a key role in exaggerating the metabolic component of the EPR evoked from hindlimb muscles with chronically-ligated femoral arteries.

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Publication Details

Journal
Acta Physiologica
Published
2026-02-16
DOI
https://doi.org/10.1111/apha.70172
Primary Topic
Heart Rate Variability and Autonomic Control
Type
article
Field-Weighted Citation Impact
0.00

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article

Myophosphorylase Knock Out Prevents the Exaggerated Exercise Pressor Reflex in Rats With Simulated Peripheral Artery Disease

Laura Anselmi, Marc P. Kaufman, Guillaume P. Ducrocq, Victor Ruiz‐Velasco
Acta Physiologica
Heart Rate Variability and Autonomic Control
article

Myophosphorylase Knock Out Prevents the Exaggerated Exercise Pressor Reflex in Rats With Simulated Peripheral Artery Disease

Laura Anselmi, Marc P. Kaufman, Guillaume P. Ducrocq, Victor Ruiz‐Velasco
article en

Abstract

Lactate and hydrogen-ions accumulation in contracting myocytes plays a key role in exaggerating the metabolic component of the EPR evoked from hindlimb muscles with chronically-ligated femoral arteries.

Acta PhysiologicaVol. 242(3)
Mitochondrie, stress oxydant et protection musculaire (FR), Penn State Milton S. Hershey Medical Center (US)
National Heart, Lung, and Blood Institute
Openalex Percentile: Top 57%
Heart Rate Variability and Autonomic Control
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