Elovl6 inhibits colorectal cancer progression through stearic acid–mediated mitochondrial fusion and metabolic reprogramming

Lipid metabolic reprogramming is a hallmark of colorectal cancer (CRC), yet the precise molecular mechanisms underlying lipid-mediated oncogenesis and the specific lipid metabolic enzymes involved remain largely elusive. Here, we identify elongation of very-long-chain fatty acid protein 6 (Elovl6) as a critical regulator in CRC progression. Clinical data reveal significant down-regulation of Elovl6 in colon cancer tissues, with low expression levels correlating with unfavorable patient prognosis. We demonstrate that Elovl6 exerts potent tumor-suppressive effects, significantly inhibiting cellular proliferation in vitro and attenuating tumor growth in vivo. Mechanistically, it maintains intestinal microbial homeostasis by preventing the expansion of opportunistic pathogens while simultaneously orchestrating metabolic reprogramming through modulation of phospholipid biosynthesis pathways. Notably, we find that stearic acid, a key Elovl6-derived metabolite, promotes mitochondrial fusion by stabilizing mitofusin 1 protein. These findings not only position Elovl6 as a promising therapeutic target but also suggest that dietary supplementation with stearic acid could represent a viable strategy for CRC prevention and treatment.

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Publication Details

Journal
Science Advances
Published
2026-02-13
DOI
https://doi.org/10.1126/sciadv.adz2892
Primary Topic
Cancer, Lipids, and Metabolism
Type
article
Field-Weighted Citation Impact
0.00
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article

Elovl6 inhibits colorectal cancer progression through stearic acid–mediated mitochondrial fusion and metabolic reprogramming

Fang Wang, Zichun Hua, Zhiqian Bi, Hongqin Zhuang et al.
Science Advances
Cancer, Lipids, and Metabolism
article

Elovl6 inhibits colorectal cancer progression through stearic acid–mediated mitochondrial fusion and metabolic reprogramming

Fang Wang, Zichun Hua, Zhiqian Bi, Hongqin Zhuang, Shuilian Fu, Shengyun Zhu, Xiaoyao Chang, Tingting Wang, Yuzhe Zhang
article en

Abstract

Lipid metabolic reprogramming is a hallmark of colorectal cancer (CRC), yet the precise molecular mechanisms underlying lipid-mediated oncogenesis and the specific lipid metabolic enzymes involved remain largely elusive. Here, we identify elongation of very-long-chain fatty acid protein 6 (Elovl6) as a critical regulator in CRC progression. Clinical data reveal significant down-regulation of Elovl6 in colon cancer tissues, with low expression levels correlating with unfavorable patient prognosis. We demonstrate that Elovl6 exerts potent tumor-suppressive effects, significantly inhibiting cellular proliferation in vitro and attenuating tumor growth in vivo. Mechanistically, it maintains intestinal microbial homeostasis by preventing the expansion of opportunistic pathogens while simultaneously orchestrating metabolic reprogramming through modulation of phospholipid biosynthesis pathways. Notably, we find that stearic acid, a key Elovl6-derived metabolite, promotes mitochondrial fusion by stabilizing mitofusin 1 protein. These findings not only position Elovl6 as a promising therapeutic target but also suggest that dietary supplementation with stearic acid could represent a viable strategy for CRC prevention and treatment.

Science AdvancesVol. 12(7)
China Pharmaceutical University (CN), Changzhou University (CN), Henan Medical University (CN), Nanjing Medical University (CN), Nanjing University (CN)
Good health and well-being
Openalex Percentile: Top 58%
Cancer, Lipids, and Metabolism
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