Multi‐Omics Dissection of the Role of Ubiquitination Modification in Post‐Stroke Neuroinflammation and Function Damage
The study identifies TRIM37 and TRIM25 as crucial regulators of neuroinflammation through ubiquitination after stroke. These findings suggest that ubiquitination plays a significant role in stroke pathology and could serve as therapeutic targets for stroke treatment.
Authors
- Dan Li (ORCID: https://orcid.org/0000-0002-4852-5485)
- Peng Yao
Institutions
- Zhejiang Chinese Medical University (CN)
- Zhejiang Hospital (CN)
- Affiliated Hangzhou First People's Hospital, Westlake University, School of Medicine (CN)
Publication Details
- Journal
- Synapse
- Published
- 2026-02-12
- DOI
- https://doi.org/10.1002/syn.70039
- Primary Topic
- Neuroinflammation and Neurodegeneration Mechanisms
- Type
- article
- Field-Weighted Citation Impact
- 0.00