Clonal Hematopoiesis in HIV and Atherosclerosis, Arterial Inflammation, and Hematopoietic Activity
Among PWH, CHIP mutations were not associated with subclinical atherosclerosis, arterial inflammation, or soluble inflammatory markers but were related to hematopoietic activity. The mechanism by which CHIP increases HIV-associated atherosclerosis may preferentially involve lymph nodes and merits additional evaluation.
Authors
- Katherine Kentoffio (ORCID: https://orcid.org/0000-0002-7755-7988)
- Hoh Rebecca
- Ahmed Tawakol
- Steven G Deeks
- A G Bick
- Matthew S Durstenfeld
- Yifei Ma
- Alexandra Evelyn Teng
- Danny Li
Institutions
- Kaiser Permanente (US)
- University of California, Los Angeles (US)
- University of California, San Francisco (US)
- Vanderbilt University (US)
- Beth Israel Deaconess Hospital (US)
- Massachusetts General Hospital (US)
Publication Details
- Journal
- medRxiv
- Published
- 2026-02-09
- DOI
- https://doi.org/10.64898/2026.02.05.26345712
- Primary Topic
- Myeloproliferative Neoplasms: Diagnosis and Treatment
- Type
- article
- Field-Weighted Citation Impact
- 0.00
Funders
- Pfizer
- Gilead Sciences
- Genentech
- National Heart, Lung, and Blood Institute