Insufficient Expression of the Autophagic Protein <scp>ATG16L1</scp> Results in Accelerated Carcinogenesis Related to an Aberrant B Cell Response

Suboptimal expression of the autophagic protein ATG16L1 results in accelerated carcinogenesis, and an altered B cell response may be one of the aggravating factors.

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Publication Details

Journal
Cancer Reports
Published
2026-02-01
DOI
https://doi.org/10.1002/cnr2.70438
Primary Topic
Autophagy in Disease and Therapy
Type
article
Field-Weighted Citation Impact
0.00
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article

Insufficient Expression of the Autophagic Protein ATG16L1 Results in Accelerated Carcinogenesis Related to an Aberrant B Cell Response

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article

Insufficient Expression of the Autophagic Protein ATG16L1 Results in Accelerated Carcinogenesis Related to an Aberrant B Cell Response

Alejandro Romero, Marisol I. González, José L. Reyes, Mónica Mendoza-Rodríguez, Luis I. Terrazas, Danielle Vannan, Miriam Rodríguez-Sosa, Diego Mateos, Daniela Mendiola, Oscar Nieto, Betsaida Ortiz, Bertus Eksteen
article en

Abstract

Suboptimal expression of the autophagic protein ATG16L1 results in accelerated carcinogenesis, and an altered B cell response may be one of the aggravating factors.

Cancer ReportsVol. 9(2)
Boston Scientific (United States) (US), Universidad Interamericana para el Desarrollo (MX), Mexfam (MX), Universidad de las Américas Puebla (MX)
Good health and well-being
Openalex Percentile: Top 62%
Autophagy in Disease and Therapy
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