The Role of Fibroblast–Epithelial Cross-Talk on the Distribution of Distinct Fibroblast Phenotypes in the Intestinal Crypt

Intestinal crypts are test tube-like structures lined with an epithelial monolayer. Under homeostasis, mitotic forces drive epithelial cells to migrate up the crypt, from the stem cell niche. As the cells migrate up the crypt, they differentiate into specialised cells. This process is regulated by morphogen gradients established by distinct populations of subepithelial fibroblasts, and recent studies suggest fibroblasts and epithelial cells have co-evolved to maintain crypt structure and function via complementary morphogen expression. We present a mathematical model of fibroblast-epithelial cross-talk, in which fibroblast and epithelial phenotypes emerge from morphogen binding to cell surface receptors. The model predicts the formation of distinct zones of mutually supporting phenotypes at different crypt heights. These findings support the idea that fibroblast and epithelial cell phenotypes are an emergent property of the crypt microenvironment. We use the model to investigate how mutations in the fibroblasts may disrupt these phenotypic zones. Our results suggest that such mutations may lead to uncontrolled epithelial cell growth and, as such, indicate how dysfunctional fibroblasts may contribute to the emergence of colorectal cancer.

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Publication Details

Journal
Bulletin of Mathematical Biology
Published
2026-02-09
DOI
https://doi.org/10.1007/s11538-025-01588-x
Primary Topic
Cancer Cells and Metastasis
Type
article
Field-Weighted Citation Impact
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article

The Role of Fibroblast–Epithelial Cross-Talk on the Distribution of Distinct Fibroblast Phenotypes in the Intestinal Crypt

George Atkinson, Hannah Byrne, S Leedham
Bulletin of Mathematical Biology
Cancer Cells and Metastasis
article

The Role of Fibroblast–Epithelial Cross-Talk on the Distribution of Distinct Fibroblast Phenotypes in the Intestinal Crypt

George Atkinson, Hannah Byrne, S Leedham
article en

Abstract

Intestinal crypts are test tube-like structures lined with an epithelial monolayer. Under homeostasis, mitotic forces drive epithelial cells to migrate up the crypt, from the stem cell niche. As the cells migrate up the crypt, they differentiate into specialised cells. This process is regulated by morphogen gradients established by distinct populations of subepithelial fibroblasts, and recent studies suggest fibroblasts and epithelial cells have co-evolved to maintain crypt structure and function via complementary morphogen expression. We present a mathematical model of fibroblast-epithelial cross-talk, in which fibroblast and epithelial phenotypes emerge from morphogen binding to cell surface receptors. The model predicts the formation of distinct zones of mutually supporting phenotypes at different crypt heights. These findings support the idea that fibroblast and epithelial cell phenotypes are an emergent property of the crypt microenvironment. We use the model to investigate how mutations in the fibroblasts may disrupt these phenotypic zones. Our results suggest that such mutations may lead to uncontrolled epithelial cell growth and, as such, indicate how dysfunctional fibroblasts may contribute to the emergence of colorectal cancer.

Bulletin of Mathematical BiologyVol. 88(3)
Centre for Human Genetics (GB), Ludwig Cancer Research (GB), Mathematical Institute of the Slovak Academy of Sciences (SK)
Openalex Percentile: Top 62%
Cancer Cells and Metastasis
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