Large-caliber choroidal arteriovenous shunt in macular neovascularization: A brief report of a novel morphological finding

This study provides direct ICGA evidence that abnormal shunts between large choroidal arteries and veins represent a novel, underrecognized etiology for MNV formation. This anatomical anomaly may act as an upstream hemodynamic driver across different choroidal backgrounds (thick or thin), potentially altering local hemodynamics and promoting neovascularization. Investigating these underlying arteriovenous relationships could improve CNV risk stratification and inform future treatment strategies.