Sleep disturbance triggers aberrant activation of vagus circuitry and induces intestinal stem cell dysfunction

Sleep disturbances are associated with pathogenesis of numerous chronic disorders, including chronic gastrointestinal diseases. However, the mechanism that transmits sleep disturbance-induced aberrant neural signaling from the brain to the gut remains elusive. We show that acute sleep deprivation (SD) impairs intestinal stem cell (ISC) function, leading to shortening of crypt-villus architecture and Paneth cell loss. We identified the dorsal motor nucleus of vagus (DMV) as the SD-sensitive central nervous system center that transmits sleep effects to the gut. SD aberrantly activates DMV neurons, driving excessive acetylcholine release from the vagus nerve into the gut. Acetylcholine triggers 5-hydroxytryptamine (5-HT) release by enterochromaffin cells and suppresses its reuptake via muscarinic receptors, thereby causing a spike in 5-HT levels. Elevated 5-HT induces excessive oxidative stress in ISCs through its receptor HTR4, promoting gut pathologies. Overall, we reveal an SD-responsive neural circuit that controls ISCs and identify therapeutic strategies for mitigating SD-related gut diseases.

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Publication Details

Journal
Cell stem cell
Published
2026-02-01
DOI
https://doi.org/10.1016/j.stem.2026.01.002
Primary Topic
Vagus Nerve Stimulation Research
Type
article
Field-Weighted Citation Impact
0.00
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article

Sleep disturbance triggers aberrant activation of vagus circuitry and induces intestinal stem cell dysfunction

M. Zhang, Yang Tian, Ning Ning, Kaichun Wu et al.
Cell stem cell
Vagus Nerve Stimulation Research
article

Sleep disturbance triggers aberrant activation of vagus circuitry and induces intestinal stem cell dysfunction

M. Zhang, Yang Tian, Ning Ning, Kaichun Wu, Di Liu, Wuqi Yang, Linlin Wang, Xiaofeng Li, Huaxing Li, Shiyang Wang, Cong Lv, Maksim V. Plikus, Liyuan Hou, Jialin Ye, Lu Yu, Feng Wang, Hanfu Zhang, Xi Wu, Zhengquan Yu
article en

Abstract

Sleep disturbances are associated with pathogenesis of numerous chronic disorders, including chronic gastrointestinal diseases. However, the mechanism that transmits sleep disturbance-induced aberrant neural signaling from the brain to the gut remains elusive. We show that acute sleep deprivation (SD) impairs intestinal stem cell (ISC) function, leading to shortening of crypt-villus architecture and Paneth cell loss. We identified the dorsal motor nucleus of vagus (DMV) as the SD-sensitive central nervous system center that transmits sleep effects to the gut. SD aberrantly activates DMV neurons, driving excessive acetylcholine release from the vagus nerve into the gut. Acetylcholine triggers 5-hydroxytryptamine (5-HT) release by enterochromaffin cells and suppresses its reuptake via muscarinic receptors, thereby causing a spike in 5-HT levels. Elevated 5-HT induces excessive oxidative stress in ISCs through its receptor HTR4, promoting gut pathologies. Overall, we reveal an SD-responsive neural circuit that controls ISCs and identify therapeutic strategies for mitigating SD-related gut diseases.

Cell stem cellVol. 33(2)
Chinese Academy of Medical Sciences & Peking Union Medical College (CN), University of California, Irvine (US), Zhengzhou University (CN), Ministry of Education (SA), Shenzhen Institutes of Advanced Technology (CN), Xijing Hospital (CN), China Agricultural University (CN), Air Force Medical University (CN)
Openalex Percentile: Top 71%
Vagus Nerve Stimulation Research
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