Cell-specific effects of acute sleep deprivation on transcriptomic signatures of non-neuronal cells in the mouse hippocampus and prefrontal cortex

Sleep is essential for maintaining cognitive, emotional, metabolic, and immune functions. Although research on sleep homeostasis is traditionally neuron-centric, increasing evidence indicates non-neuronal cells also play critical roles. In this study, we performed transcriptomic analyses of non-neuronal nuclei from the hippocampus and prefrontal cortex of mice subjected to acute sleep deprivation (SD). We found that acute SD induces robust, cell-type- and region-specific transcriptional reprogramming in astrocytes and oligodendrocytes. The most pronounced changes occurred in astrocytes, including downregulation of cholesterol biosynthesis genes in both brain regions, accompanied by region-specific and opposing regulation of genes involved in mitochondrial function and neurodegeneration-related pathways. Notably, genes associated with primary cilia were selectively induced in cortical astrocytes. In oligodendrocytes, acute SD led to downregulation of genes encoding cell-adhesion molecules. Together, these findings provide molecular evidence that non-neuronal cells actively contribute to sleep regulation and suggest novel potential mechanisms that broaden our understanding of sleep homeostasis.

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Publication Details

Journal
bioRxiv (Cold Spring Harbor Laboratory)
Published
2026-02-03
DOI
https://doi.org/10.64898/2026.01.29.702391
Primary Topic
Sleep and Wakefulness Research
Type
article
Field-Weighted Citation Impact
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article

Cell-specific effects of acute sleep deprivation on transcriptomic signatures of non-neuronal cells in the mouse hippocampus and prefrontal cortex

Ted M. Abel, Yann Vanrobaeys, Junko Kasuya, Yuxuan Wang et al.
bioRxiv (Cold Spring Harbor Laboratory)
Sleep and Wakefulness Research
article

Cell-specific effects of acute sleep deprivation on transcriptomic signatures of non-neuronal cells in the mouse hippocampus and prefrontal cortex

Ted M. Abel, Yann Vanrobaeys, Junko Kasuya, Yuxuan Wang, Marcos G. Frank
article en

Abstract

Sleep is essential for maintaining cognitive, emotional, metabolic, and immune functions. Although research on sleep homeostasis is traditionally neuron-centric, increasing evidence indicates non-neuronal cells also play critical roles. In this study, we performed transcriptomic analyses of non-neuronal nuclei from the hippocampus and prefrontal cortex of mice subjected to acute sleep deprivation (SD). We found that acute SD induces robust, cell-type- and region-specific transcriptional reprogramming in astrocytes and oligodendrocytes. The most pronounced changes occurred in astrocytes, including downregulation of cholesterol biosynthesis genes in both brain regions, accompanied by region-specific and opposing regulation of genes involved in mitochondrial function and neurodegeneration-related pathways. Notably, genes associated with primary cilia were selectively induced in cortical astrocytes. In oligodendrocytes, acute SD led to downregulation of genes encoding cell-adhesion molecules. Together, these findings provide molecular evidence that non-neuronal cells actively contribute to sleep regulation and suggest novel potential mechanisms that broaden our understanding of sleep homeostasis.

bioRxiv (Cold Spring Harbor Laboratory)
University of Iowa (US), Washington State University Spokane (US), Center for Neurosciences (US), Massachusetts Institute of Technology (US)
Openalex Percentile: Top 69%
Sleep and Wakefulness Research
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