Anoctamin 5 mutation leads to abnormal bone homeostasis of GDD by regulating AMPK-dependent glucose metabolism

We highlighted the critical role of glucose metabolic distemperedness mediated by AMPK activation in GDD, which developing a potential therapeutic strategy targeted for treatment of GDD.

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Publication Details

Journal
Frontiers in Endocrinology
Published
2026-02-04
DOI
https://doi.org/10.3389/fendo.2026.1703491
Primary Topic
Bone Metabolism and Diseases
Type
article
Field-Weighted Citation Impact
0.00
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article

Anoctamin 5 mutation leads to abnormal bone homeostasis of GDD by regulating AMPK-dependent glucose metabolism

Xu Han, Liqi Lin, Shengnan Wang, Hongyu Li et al.
Frontiers in Endocrinology
Bone Metabolism and Diseases
article

Anoctamin 5 mutation leads to abnormal bone homeostasis of GDD by regulating AMPK-dependent glucose metabolism

Xu Han, Liqi Lin, Shengnan Wang, Hongyu Li, Rui Dong, Ying Hu, Kaiwen Sun, Shuai Zhang, Mingyue Zhang
article en

Abstract

We highlighted the critical role of glucose metabolic distemperedness mediated by AMPK activation in GDD, which developing a potential therapeutic strategy targeted for treatment of GDD.

Frontiers in EndocrinologyVol. 17
Capital Medical University (CN), Beijing Obstetrics and Gynecology Hospital (CN)
Openalex Percentile: Top 69%
Bone Metabolism and Diseases
DOIOpen Access PDFOpenAlex
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