FSTL1 contribute to aggressive clinical behavior in DLBCL may by activating the DIP2A/ICAM-1-mediated adhesion mechanism
Our findings indicate that elevated FSTL1 levels may contribute to advanced clinical characteristics and worse outcomes in DLBCL. FSTL1 contributes to drug resistance likely through DIP2a/ICAM-1-mediated adhesion mechanism.
Authors
- Cunde Xiao (ORCID: https://orcid.org/0000-0002-1706-4794)
- X. Zhang
- Yao Liu (ORCID: https://orcid.org/0000-0001-5572-723X)
- Yixing Zhou
- Xiping Xiping Liang
- W. Zhou
- Chaoyu Wang
- Xiaoqing Xie
Institutions
- Chongqing Cancer Hospital (CN)
Publication Details
- Journal
- Biomedicine & Pharmacotherapy
- Published
- 2026-01-29
- DOI
- https://doi.org/10.1016/j.biopha.2026.119071
- Primary Topic
- Cell Adhesion Molecules Research
- Type
- article
- Field-Weighted Citation Impact
- 0.00