Proteomics Profiling Reveals Circulating Biomarkers and Dysregulated Pathways in Transthyretin Amyloid Cardiomyopathy

This study reveals a distinct proteomic profile of ATTR-CM compared with controls with HF, and elucidates both novel and known signaling pathways that are differentially regulated in ATTR-CM.

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Publication Details

Journal
Circulation Heart Failure
Published
2026-01-22
DOI
https://doi.org/10.1161/circheartfailure.125.013220
Primary Topic
Amyloidosis: Diagnosis, Treatment, Outcomes
Type
article
Field-Weighted Citation Impact
0.00
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article

Proteomics Profiling Reveals Circulating Biomarkers and Dysregulated Pathways in Transthyretin Amyloid Cardiomyopathy

Ani Nalbandian, Keitaro Akita, Yuichi J. Shimada, Alfonsina Mirabal Santos et al.
Circulation Heart Failure
Amyloidosis: Diagnosis, Treatment, Outcomes
article

Proteomics Profiling Reveals Circulating Biomarkers and Dysregulated Pathways in Transthyretin Amyloid Cardiomyopathy

Ani Nalbandian, Keitaro Akita, Yuichi J. Shimada, Alfonsina Mirabal Santos, Ree Lu, Mathew S. Maurer, Sergio Teruya, Dimitrios Bampatsias
article en

Abstract

This study reveals a distinct proteomic profile of ATTR-CM compared with controls with HF, and elucidates both novel and known signaling pathways that are differentially regulated in ATTR-CM.

Circulation Heart FailureVol. 19(2)
Columbia University Irving Medical Center (US)
Reduced inequalities
Openalex Percentile: Top 82%
Amyloidosis: Diagnosis, Treatment, Outcomes
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