Nicotinamide adenine dinucleotide inhibits the production of reactive oxygen species and myocardial cell pyroptosis caused by hypoxia/re-oxygenation injury

These results indicate that NAD<sup>+</sup> supplementation may serve as a promising therapeutic strategy for I/R injury.

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Publication Details

Journal
World Journal of Cardiology
Published
2026-01-15
DOI
https://doi.org/10.4330/wjc.v18.i1.114108
Primary Topic
Inflammasome and immune disorders
Type
article
Field-Weighted Citation Impact
0.00
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article

Nicotinamide adenine dinucleotide inhibits the production of reactive oxygen species and myocardial cell pyroptosis caused by hypoxia/re-oxygenation injury

Zuowei Pei, Yijun Tu, Shuang Lin Dong, Chang Liu et al.
World Journal of Cardiology
Inflammasome and immune disorders
article

Nicotinamide adenine dinucleotide inhibits the production of reactive oxygen species and myocardial cell pyroptosis caused by hypoxia/re-oxygenation injury

Zuowei Pei, Yijun Tu, Shuang Lin Dong, Chang Liu, Shan Gao, Yun-Qi Liu, Yu-Jie Liu
article en

Abstract

These results indicate that NAD<sup>+</sup> supplementation may serve as a promising therapeutic strategy for I/R injury.

World Journal of CardiologyVol. 18(1)
Dalian Municipal Central Hospital (CN)
Openalex Percentile: Top 87%
Inflammasome and immune disorders
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Nicotinamide adenine dinucleotide inhibits the production of reactive oxygen species and myocardial cell pyroptosis caused by hypoxia/re-oxygenation injury — Zuowei Pei, Yijun Tu, et al. · World Journal of Cardiology (2026) | TGRS Research Map | TGRS